interneurons


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in·ter·neu·rons

(intĕr-nū'ronz),
Combinations or groups of neurons between sensory and motor neurons that govern coordinated activity.
Farlex Partner Medical Dictionary © Farlex 2012

in·ter·neu·rons

(in'tĕr-nūr'onz)
Combinations or groups of neurons between sensory and motor neurons that govern coordinated activity.
Synonym(s): interneurones.
Medical Dictionary for the Health Professions and Nursing © Farlex 2012

in·ter·neu·rons

(in'tĕr-nūr'onz)
Combinations or groups of neurons between sensory and motor neurons that govern coordinated activity.
Medical Dictionary for the Dental Professions © Farlex 2012
References in periodicals archive ?
Obesity-induced type 2 diabetes impairs neurological recovery after stroke in correlation with decreased neurogenesis and persistent atrophy of parvalbumin-positive interneurons
He will examine brain tissue donated by people who died with DLB, focusing on specialised cells called interneurons. Interneurons co-ordinate the activity of groups of brain cells that are necessary for functions like learning and memory.
N-methyl-D-aspartate receptors localized on the GABA interneuron modulate GABAergic activity.
Rapid GABAergic IPSPs elicited by B40 in interneurons and motor neurons also bias motor programs toward ingestion by coordinating closure of the radula with its retraction phase, resulting in an inward displacement of food toward the esophagus (Jing et al., 2003).
They were also positive for GABA and other markers of specialized subclasses of inhibitory interneurons, which was the goal.
The potential effects of male exposure in the response of the L3 prothoracic auditory interneuron are unknown.
Interneurons control complex networks between neurons, allowing them to send signals to one another in a harmonized way.
A previous study found that ASSRs induced energy in the gamma band was significantly lower in schizophrenia patients than in healthy subjects.[7] A follow-up study suggested that it might be linked with volume changes in GABAergic interneurons and pyramidal cells[8], leading to excitation and inhibition deficits.
A number of mechanisms have been suggested for central sensitization, among which there is strongest evidence for the dysfunction of GABAergic interneurons and their role in modifying synaptic transmission of pain signaling pathways in response to PNI [11-13].
Incoming glutamatergic perforant path fibers form excitatory synapses on both the principal glutamatergic granule cells and neighboring GABAergic interneurons, for example, basket cells [28].
Normally, when the circuit is turned on, neurons of the prefrontal cortex activate certain neurons called high-firing interneurons, which then suppress striosome activity.