The primary inotropic agents
in our study were digitalis and dopamine.
Patient's capillary withdrawal time was prolonged (>5 sec) and were given inotropic agents
, vasopressor support and saline challange twice a day (20 cc/kg) Her blood gases were; pH 6,56, pCO2: 104 mmHg, HCO3:9,2 mmol/L, Be:-31,6, lactat: 16,32 mmol/L at her admission.
The control group was given continuous low flow oxygen inhalation and inotropic agents
, and their cardiac stress was also reduced.
Suggested escalating levels of treatment include no treatment for an abnormality on radiography alone; nasal supplemental oxygen for mild hypoxemia; continuous positive airway pressure through face mask for moderate hypoxemia and intubation, mechanical ventilation, and positive end-expiratory pressure for severe hypoxemia; and volume replacement and inotropic agents
for hypotension with low cardiac output.
The presence and the severity of cardiac failure may require preoperative management with inotropic agents
Pimobendan is a phosphodiesterase III inhibitor and calcium channel sensitizer causing a inodilating effect, at reduced myocardial oxygen consumption energy expenditure when compared to other positive inotropic agents
as indicated by Fuentes, 2004 and Atkinson et al.
and broad-spectrum antibiotics were administered under the situation of severe sepsis.
While being on mechanical ventilation, he was given antibiotics, anticonvulsants, inotropic agents
, oxygen supplementation, albumin infusion and other supportive management.
Temporary therapeutic measures include rapid fluid infusion, administration of positive inotropic agents
, and pericardiocentesis.
New positive inotropic agents
with higher safety margins and stronger effects on cardiac contractility have been introduced in human and veterinary cardiology during the last 20 years (3) and their use must be explored in the avian patient.
10] Acute Physiology and Chronic Health Evaluation (APACHE) II scores,  the presence of bowel sounds and co-administration of sedatives and inotropic agents
were documented on enrolment.
The study retrospectively reviewed the medical records of 56 consecutive patients who presented to Tabba Heart Institute, Karachi, from June 2009 to June 2011 with STEMI (defined as electrocardiographic evidence of ST segment elevation of Greater than 1mm in Greater than 2 contiguous leads or new left bundle branch block) who developed cardiogenic shock (defined as presence of systolic blood pressure Less than 90mmHg or need of parenteral inotropic agents
with evidence of systemic hypoperfusion like cold clammy skin, cerebral obtundation) within 36 hours of the onset of STEMI, and underwent PPCI within 18 hours of the onset of the shock.