infarct


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infarct

 [in´fahrkt]
a localized area of ischemic necrosis produced by anoxia following occlusion of the arterial supply or the venous drainage of the tissue, organ, or part.
anemic infarct one due to sudden interruption of arterial circulation to the area.
hemorrhagic infarct one that is red owing to oozing of erythrocytes into the injured area.

in·farct

(in'farkt),
An area of necrosis resulting from a sudden insufficiency of arterial or venous blood supply.
Synonym(s): infarction (2)
[L. in-farcio, pp. -fartus (-ctus, an incorrect form), to stuff into]

infarct

(ĭn′färkt′, ĭn-färkt′)
n.
An area of tissue that undergoes necrosis as a result of obstruction of local blood supply, as by a thrombus or embolus.

in·farct′ed adj.

infarct

Pathology Dead/necrotic tissue. See Acute myocardial infarct, Anemic infarct, Lacunar infarct, Myocardial infarct, Non-Q-wave infarct, Pseudoinfarct, Q wave infarct, Red infarct, Reperfusion-eligible acute myocardial infarct, Watershed infarct, White infarct. Cf Infarction.

in·farct

(in'fahrkt)
An area of necrosis resulting from a sudden insufficiency of arterial or venous blood supply.
Synonym(s): infarction (2) .

infarct

A volume of dead tissue lying within living tissue, the death being caused by local loss of blood supply. Infarcted tissue swells and becomes firm, and blood vessels around an infarct widen. Plasma and blood may pass into the infarct, increasing the swelling. Later the infarct becomes pale and shrinks and soon it is replaced by fibrous tissue and is converted into a scar which is usually at least as strong as the original tissue. Function is, of course, lost.

Infarct

Death of tissue due to shutting off the blood supply.
Mentioned in: Tetralogy of Fallot

in·farc·tion

(in-fahrk'shŭn)
Area of tissue necrosis caused by impaired arterial or venous blood supply due to mechanical factors (e.g., emboli, thrombi) or to blood pressure alterations.
Synonym(s): infarct.
References in periodicals archive ?
In the infarct area, the spatial distribution of Iba-1 signals was correlated with those of TNF-[alpha] (Figures 6(a)-6(h)), although the two signals were never overlapped, whether in the infarct area (Figures 6(a)-6(d)) or the intact striatum and corpus callosum (Figures 6(e)-6(h)).
Steinling, "Residual deficit of verbal recall in a left internal cerebral vein infarct," Revue Neurologique, vol.
We describe an 81-year-old female patient with cortical infarct who presented with sudden onset isolated foot drop that easily interfered with peripheric nerve lesions.
Infarcts with diameter ranging from 1.4 mm to 15.0 mm were observed in 23/27 (85.0%) patients; these were distributed in the subcortical white matter (42.3%), basal ganglia (30.2%), thalamus (16.4%), brain stein (10.0%), cerebellum (1.0%), and the cortex [Figure 3]b and [Figure 3]c.
Another reason for a lack of association between stroke patterns and AF in both the Find-AF and the CRYSTAL-AF study could be the inclusion of patients with small strokes and low National Institutes of Health Stroke Scale (NIHSS) (median NIHSS in the CRYSTAL-AF was <2 and in our study 3), as there is a known association between large infarct volumes and high NIHSS in patients with cardioembolic strokes caused by AF [17].
Type III collagen was the main type of collagen in Infarct Zone of the unloading group.
Caption: FIGURE 1: Computed Tomography (CT) of the abdomen done at admission with intravenous and oral contrast: the yellow arrow shows a high-density fluid filled large defect in the superior aspect of the spleen consistent with splenic infarct.
This finding led to the second trial, which concentrated on infarcts from the LAD in 301 patients.
AOP infarcts are unique in their bilateral, symmetric nature and can be mistaken for a systemic, rather than a vascular process.
Higher average diastolic blood pressure was also related to brain infarct lesions.