hypoxic stress


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hypoxic stress

). Note: the strain can be either the cause or the effect. Syn. stress. See annealing; asthenopia; hypoxia; polariscope.
References in periodicals archive ?
On long-term hypoxic stress, there is an energy deprivation in tumor cell which results in elevated AMP/ATP ratio, leading to the activation of AMPK, a major regulator of cellular energy homeostasis.
Hypoxic stress and IL-7 gene overexpression enhance the fusion potential of rat bone marrow mesenchymal stem cells with bovine renal epithelial cells.
teleta can use anaerobic respiratory pathways at different stages of development when faced with hypoxic stress (e.
He said tumor cells grow really fast, but the blood vessels that feed them oxygen cannot grow fast enough, so tumor cells become hypoxic, adding that the tumor cells have to develop some sort of mechanism to survive under hypoxic stress, and that's HIF-1 alpha.
Here we hypothesize that exposure of the prostatic epithelial tissue to lipopolysaccharide from gram negative bacteria and hypoxic stress may induce prostate cancer associated cellular behaviors in mice, with important implication in human.
virginica adults had similar aerobic metabolic rates; however, differences may exist in the metabolic pathways and rate used during hypoxic stress.
Indeed, nuclear HIF expression has been detected in medullary structures following the administration of radiocontrast media [10], indicating the induction of cellular hypoxic stress response by contrast agents.
4) A more recent study by Papas suggests a higher threshold is necessary to avoid zero hypoxic stress (125x[10.
Protein carbonyls as a biomarker of foetal-neonatal hypoxic stress.
after the "hot" or inflammatory phase) and as such complements the body's natural rescue mechanism for those cells that face hypoxic stress (i.
The intermittent hypoxia of sleep apnea and the continuous hypoxia of altitude are conditions of hypoxic stress that are known to modulate glucose and insulin homeostasis.
Cancer metabolism research seeks to explain why cancer cells use energy differently to normal cells in order to survive and grow, particularly under the conditions of nutrient and hypoxic stress faced by rapidly growing tumours.