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11] Renal tubular function is also affected leading to the manifestation of hypocalciuria.
The etiology of hypocalciuria in preeclamptics remains obscure, but the study by Taufield et al.
In our study, hypocalciuria is significantly increased in pregnant women with preeclampsia although CCR is not significant.
The laboratory tests result exhibited obviously low urine specific gravity, increased urine pH, hypokalemia, hypochloremia, hyperkaluria, hypocalciuria, metabolic alkalosis, increased plasma renin activity and angiotensin II, and low blood pressure.
Key laboratory findings include hypokalemia, metabolic alkalosis, hypomagnesemia, and hypocalciuria (3-11).
Hypocalciuria is another biochemical hallmark of GS that can be explained by increased passive calcium absorption secondary to extracellular volume contraction.
This might be responsible for hypocalciuria in pre-eclampsia.
Hence they suggested that, hypocalciuria in preeclampsia is independent of renal function and reflects a complex alteration in calcium homeostasis at the cellular level and can be used for predicting preeclampsia.
Diagnostic features include a low urinary pH, low urinary volumes, hyperoxaluria, hypocalciuria and hypocitraturia.