hyperglycemia


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Related to hyperglycemia: hypoglycemia

hyperglycemia

 [hi″per-gli-se´me-ah]
excess of glucose in the blood; see also diabetes mellitus.

hy·per·gly·ce·mi·a

(hī'pĕr-glī-sē'mē-ă),
An abnormally high concentration of glucose in the circulating blood, seen especially in patients with diabetes mellitus.
Synonym(s): hyperglycosemia
[hyper- + G. glykys, sweet, + haima, blood]

hyperglycemia

(hī′pər-glī-sē′mē-ə)
n.
The presence of an abnormally high concentration of glucose in the blood.

hy′per·gly·ce′mic (-mĭk) adj.

hyperglycemia

Metabolism An abnormal ↑ in serum glucose, most commonly due to DM. See Diabetes mellitus, Glucose tolerance test, Hyperglycinemia.

hy·per·gly·ce·mi·a

(hī'pĕr-glī-sē'mē-ă)
An abnormally high concentration of glucose in the blood, a feature of diabetes mellitus.
Synonym(s): hyperglycaemia.
[hyper- + G. glykys, sweet, + haima, blood]

Hyperglycemia

Condition characterized by excessively high levels of glucose in the blood, and occurs when the body does not have enough insulin or cannot use the insulin it does have to turn glucose into energy. Hyperglycemia is often indicative of diabetes that is out of control.

hy·per·gly·ce·mi·a

(hī'pĕr-glī-sē'mē-ă)
Abnormally high concentration of glucose in the circulating blood, seen in diabetes mellitus.
Synonym(s): hyperglycaemia.
[hyper- + G. glykys, sweet, + haima, blood]
References in periodicals archive ?
The study, which assessed nearly 15,000 users, found meaningful differences in hyperglycemia post-meal associated with different glucose levels and trend arrows at meals.
Neurological dysfunction associated with nonketotic hyperglycemia. Arch Neurol 1968;19(5):525-534.
Alterations in glucose homeostasis in the pediatric intensive care unit: Hyperglycemia and glucose variability are associated with increased mortality and morbidity.
(1996) Chorea-ballismus with nonketotic hyperglycemia in primary diabetes mellitus.
Chorea associated with non-ketotic hyperglycemia and hyperintensity basal ganglia lesions on T1-weighted brain MRI study: a meta-analysis of 53 cases including four present cases.
Conclusion: Steroid induce hyperglycemia occurred in 18.7% of the study population.
The first survey showed that 97/192 (51%) intensive care physicians did not use glucose solution for fluid resuscitation; only 142/188 (76%) respondents paid much attention to suppressing the BG variability; 104/192 (54%) respondents routinely detected BG levels every 4 h in critically ill patients; 159/189 (84%) respondents mixed glucose insulin according to 4-6 g glucose per 1 unit insulin when infusing glucose solution; and 53/190 (28%) respondents determined the dose of regular insulin for treating hyperglycemia based on their experiences.
This time course of events demonstrates how treatment of hyperglycemia can adequately treat and lead to complete resolution of hemichorea-hemiballismic movements in patients with CHBG.
But, the mechanisms underlying these associations are not fully understood, and whether hyperglycemia is causally related to adverse events after acute MI is unclear.
In addition, prolonged hyperglycemia has been shown to induce A-FABP expression in mesangial cells and trigger the release of proinflammatory cytokines [22].
One of the key elements in DM pathology is hyperglycemia. Hyperglycemia was shown to disturb the cell machinery yielding distorted biological responses.
Our patient with hyperglycemia and dysmorphic features had a deletion of 7.23 Mb comprising the region 7p13-p12.1, with involvement of 39 Online Mendelian Inheritance in Man genes, including glucokinase associated with maturity-onset diabetes of the young, CCM2 associated with type 2 cerebral cavernous malformations, insulin-like growth factors binding protein-3 associated with decreased postnatal growth, and oxoglutarate dehydrogenase associated with alpha-ketoglutarate dehydrogenase deficiency (short stature, hypotonia, cognitive impairment, and movement abnormalities).