Let a be the homozygotic effect of a mutation on Y (i.e., the expected difference for the trait value between wild and mutant homozygotes).
For the sake of comparison with results from dataset 1, chromosomal effects for this homozygote viability measurement will be interpreted as the sum of the homozygotic effects a of the mutations carried.
Homozygotic viability effects are interpreted as in dataset 2.
Expected homozygotic effect of mutations and probability of having positive effect, both obtained from the minimum-distance estimates in Table 1.
Thus, irrespectively of estimates being conditioned or not conditioned to [Lambda] = 1, about 95% of the mutational variance is due to about 1% of the molecular mutations with an absolute homozygotic effect on viability greater than 0.1 for the Mukai et al.
Thus, homozygotic lethals represent a special mutational class.
In its simplest form, this model postulates that individuals homozygotic
for the D allele (DD genotype) are all right-handed, those homozygotic
for the C allele (CC genotype) are equally likely to be left- or right-handed, while heterozygotes (DC genotype) have a .75 probability of being right-handed.
To illustrate the extent of such constraints, we obtain some simple predictions for a trait undergoing symmetric stabilizing selection of the strength estimated in this experiment, and additive mutations at a per locus rate [Micro], with homozygotic
effects a symmetrically distributed around zero, producing the [[Sigma].sup.2.sub.m] obtained here.
Whether specimens have a homozygotic
or heterozygotic allele of the [(A).sub.10] repeat can be shown more clearly by the number of specific peaks in an electropherogram.
This population structure would be evidenced by a higher proportion of homozygotic
individuals than expected by chance (Wahlund Effect).
patterns may also be observed if parental taxa contributed identical alleles at homologous loci.
Our results suggest that only the two homozygotic
peaks are stable equilibria.