* Laboratory-confirmed criteria: serologic evidence of a fourfold change in IgG-specific antibody titer reactive with spotted fever
group antigen by indirect IFA between paired serum specimens (one taken in the first week of illness and a second 2-4 weeks later), or by polymerase chain reaction, immunohistochemistry, or cell culture.
Thus, thrombocytopenia or hypercoagulable state is not associated with any blood
group antigen. However larger sample size and more rigorous analysis such as von Willebrand factor or blood serotonin level can shade more light in this area of research.
Compared with the histo-blood
group antigen (HBGA) binding surface of strain SMV, the GII.2 strains in this study had 15 aa mutations in VP1, including 8 aa mutations at residues 335-349 around the HBGA binding site I in the P2 domain, although the conserved set of residues (Asn352, Arg353, Asp382, and Gly445) required for binding the fucose moiety of HBGAs in SMV was unchanged.
ACKR1 was identified as a blood
group antigen that was expressed on the human red blood cell surface in 1950 (22).
Expressed mainly in the intestinal tissue, the alpha-(1,2)-fucosyltransferase FUT2 plays an important role in the formation of the ABH tissue blood
group antigen in the intestine.
used long-range surface plasmon waveguides for capturing only blood
group antigen A on RBCs by immobilized Anti-A IgG via no comparative studies with Anti-B [29].
Prevalence of Diego blood
group antigen and the antibody in three ethnic population groups in Klang valley of Malaysia.
Ten per cent of the white population lack the Lewis blood
group antigen and are unable to produce CA 19-9, even in the presence of malignant disease.M High levels of CA 19-9 were subsequently reported in other gastrointestinal malignancies, including pancreas, bile duct, oesophageal and gastric cancer, as well as non-gastrointestinal tumours.[5] Increased levels have also been observed in non-malignant causes of cholestatic jaundice, particularly chronic pancreatitis and choledocholithiasis.
Lowe, "Molecular cloning, sequence, and expression of a human GDP-L-fucose: [beta]-D-galactoside 2-[alpha]-L-fucosyltransferase cDNA that can form the H blood
group antigen," Proceedings of the National Academy of Sciences of the United States of America, vol.
The only type of glycan that interacted with VP8* was type A histo-blood
group antigen, he said.
In contrast, the A blood
group antigen, comprised of blood types A and AB, appears to be protective against diminished ovarian reserve.
The expression of this sialylated [Le.sup.a] blood
group antigen (sialylated lactoN-fucopentoeose II ganglioside) is required for the expression of CA 19-9 and hence [Lea.sup.a-b-] patients do not express the antigen and can present as false negatives (Steinberg, 1990).