Identification of an N-capping box that affects the alpha 6-helix propensity in glutathione S-transferase
superfamily proteins: a role for an invariant aspartic residue.
Another mechanism is the production of FosA, a glutathione S-transferase
that inactivates fosfomycin by addition of a glutathione residue.
Background: Several studies concerning the association between glutathione S-transferase
P1 (GSTP1) Ile105Val polymorphism and male infertility risk have reported controversial findings.
There were 12 glutathione S-transferase
(GST) genes up-regulated.
Research shows that natural compounds can reverse drug resistance by inhibition of P-glycoprotein, inhibition of glutathione S-transferase
drug detoxification system and inhibition of heat-shock proteins.
Among the identified proteins, we observed decreases in the expression of phospholipid hydroperoxide glutathione peroxidase (PHGPX); ATP synthase subunit O, mitochondrial (ATP5O); glutathione S-transferase
Mu 5 (GSTM5), mitochondrial NADH dehydrogenase [ubiquinone] 1 alpha subcomplex subunit 10 (NDUFA10).
A systemic review of glutathione S-transferase
P1 Ile105Val polymorphism and colorectal cancer risk.
The effect of environmental parameters on growth, cholinesterase activity and glutathione S-transferase
activity in the earthworm Aporectodea caliginosa.
2) In parallel, cells have a comprehensive system of antioxidant defences to prevent free radical formation, and the glutathione S-transferase
(GST) multifamily of enzymes constitutes an important antioxidant system for ROS detoxification and protection against toxicity.
Association between glutathione S-transferase
pi polymorphisms and survival in patients with advanced nonsmall cell lung carcinoma.
The synergists used in these experiments were piperonyl butoxide (PBO), a mixed-function microsomal oxidase inhibitor; S,S,S-tributyl phosphorotrithionate (DEF), a putative inhibitor of esterases; triphenyl phosphate (TPP), an inhibitor of carboxylesterase; diethyl maleate (DEM), a glutathione S-transferase
inhibitor (Grant et al.
M1*null genotype but not myeloperoxidase promoter G-463A polymorphism is associated with higher susceptibility to endometriosis.