In the present study, degenerative changes in columnar epithelial cells, vacuolated basal region, brush border disappearance, top plate thinning, distortion in gastric glands
and fusion of submucosa with mucosal folds were frequently observed pathological alterations in stomach of A.
Quantitative immunohistomorphological microscopic analysis was performed by counting the GLP-1R immunoreactive and nonimmunoreactive glandular cells using Mac Biophotonic ImageJ (a Java program inspired by National Institute of Mental Health, Bethesda, Maryland, USA) in a minimum of 10 well-oriented high-quality longitudinal cuts of gastric glands
. Frequency of GLP-1R immunoreactive cells was determined separately in neck, mid, and bottom gland areas.
A, Multiphoton microscopy image shows architecture of typical columnar epithelium in gastric cardia with gastric glands
([less than or equal to] 60 m depth).
The latter revealed that cotton pellet implantation caused mild degeneration changes in gastric glands
. Probably due to immune mediated mechanism triggered by foreign implantation, doses of C.
The determined measurements were the height for the mucosal projection and the thickness for lamina propria, inner circular muscle, outer longitudinal muscle, and gastric gland
There were many simple and tubular gastric glands
, consisting of cells called oxintopeptics, secreting H + and Cl- and pepsinogen (Figure 4A).
Helicobacter pylori detected deep in gastric glands
: an ultrastructural quantitative study.
Pathology showed crypt dropout of the gastric glands
, consistent with mild GVHD.
have not been observed in this specimen.
The tissues also showed normal gastro - duodenal junction and normal gastric glands
. The animals of remaining groups were induced for ulcer by giving 1 ml of 100% ethanol after keeping them on fasting for 48 hrs.
Gastrintestinal endocrine cells were dispersed throughout the epithelia and gastric glands
of the alimentary tract.
It increases the pH around the parasites so as to avoid the action of gastric acids or pepsin on its cuticle [49, 80, 106], so there is an inhibition in the transformation of pepsinogen, produced in the principal cells of the gastric glands
, to pepsin.