aeruginosa produces a number of virulence factors which help in invasion and damage to host tissues.16 The expression of most of these virulence factors are regulated by two component transcriptional regulatory system and quorum sensing which are essential for the survival and growth of bacteria in the host.4 The most crucial virulence factors are alginate, elastase, exotoxin A, phospholipase and exoenzyme
S which are regulated through proper signalling mechanisms.17 When the bacteria are high in number, P.
However, further studies are necessary to understand the role of individual bacteria and the exoenzymes
in bloom regulation.
Accumulation of Mycelium Biomass andProteolytic Exoenzymes
ADP-Ribosylation and Subcellular Redistribution of Rac1 by Exoenzyme
VPA1327 encoding an exoenzyme
T revealed 64% (over 233 amino acids) and 46% (over 173 amino acids) with exoenzyme
T of Providencia alcalifaciens and Pseudomonas aeruginosa, respectively.
A variety of genes are under the quorum sensing control regulon many of which modulate bacterial virulence including genes involved in exoenzyme
production and biofilm formation [15-17].
(6) Other virulence factors can cause pulmonary damage by different mechanisms such as Exoenzyme
S, Exotoxin A, Elastase and Phospholipases.
S (ExoS) is a bifunctional protein secreted by Pseudomonas aeruginosa via the bacterial type III secretory (TTS) process.
Recently it has been shown that various other autoinducer molecules regulate the production of exoenzyme
virulence determinants in Pseudomonas aeruginosa and Erwinia carotovora (Jones et al., 1993).
: alkaline protease , elastase , exotoxin A, exoenzyme
S and hemolysin are produced during the course of clinical infection and contribute for the development of infections in animal models [10,11,14].
albicans many virulence factors, such as germ tube formation, exoenzyme
production, and phenotypic switching (10).
ENVIRONMENTAL EFFECTS ON A PROTEASE EXOENZYME
PRODUCED BY A HALOPHILIC BACTERIUM ISOLATED FROM AN INLAND SALT SPRING.