exertional rhabdomyolysis

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ex·er·tion·al rhab·do·my·ol·y·sis

rhabdomyolysis produced in those susceptible by muscular exercise.
Episodic haemoglobinuria due to haemolysis caused by repeated mechanical injury to red cells that travel through small vessels overlying the bones of hands and feet in long-distance marching—soldiers, marathon running, calisthenics, karate
Lab Myoglobinuria, proteinuria, increased BUN, increased enzymes—e.g., creatinine phosphokinase, increased lactic acid


emanating from or pertaining to exertion.

exertional rhabdomyolysis
occurs as an acute recumbency or immobility state when muscle masses disrupt during exercise. Occurs as equine paralytic myoglobinuria in horses after unaccustomed exercise while on a heavy carbohydrate diet, in sheep and cattle after violent physical exercise, in racing greyhounds and as capture or transport myopathy in deer and other wildlife. Called also azoturia, Monday morning disease, tying up syndrome, capture myopathy.


disintegration of striated muscle fibers with excretion of myoglobin in the urine.

equine rhabdomyolysis
see paralytic myoglobinuria.
exertional rhabdomyolysis
see exertional rhabdomyolysis.
transient exertional rhabdomyolysis
References in periodicals archive ?
8 percent of the soldiers without sickle cell trait who developed exertional rhabdomyolysis, just over 1 percent of black soldiers with the trait developed the condition, Kurina said.
We report the case of a young, active duty SCT positive African American male presenting with exertional rhabdomyolysis complicated by myoglobinuric renal failure requiring hemodialysis.
Do exercise specialists have a legal duty to protect those under their supervision from the risk of exertional rhabdomyolysis as a consequence of overload during exercise?
However, there is also concern that athletes with SCT have an elevated risk of exercise-related sudden death secondary to exertional heat illness or acute exertional rhabdomyolysis (Eichner 2010).
31) There is evidence that persons with sickle cell trait have increased susceptibility to exertional rhabdomyolysis, with the potential for renal failure and death.
Effect of ration and exercise on plasma creatina kinase activity and lactate concentration in Thoroughbred horses with recurrent exertional rhabdomyolysis.
Exertional rhabdomyolysis is often seen following strenuous muscular exercise and is a response to intensive and severe exercise.
The most common etiology of exertional rhabdomyolysis is strenuous physical exercise.
During the summer and fall of 1988, outbreaks of exertional rhabdomyolysis (the breakdown of muscle fiber) with renal impairment occurred in New York and Massachusetts among candidates or trainees for public safety positions.
According to the autopsy, Plancher died as a result of "dysrhythmia brought on by exertional rhabdomyolysis with sickle-cell trait.