overactivity reflects deterioration in clinical status and cardiorespiratory reflex control in chronic heart failure.
The potential candidates underlying the reduced sympathetic nerve activity by AET include afferent autonomic control coordinated by arterial baroreceptors, cardiopulmonary receptors, ergoreceptors
, and chemoreceptors [143-145].
The mechanism may relate to the release of ergoreceptors
, afferent nerve activity and increased [beta]-endorphin production/release that are stimulated by needle insertion into skin and muscle.
They activate what are known as ergoreceptors
in the muscle that signalize to the body that the muscle cannot sustain the workload.