Small intestinal development in the young chick: crypt formation and enterocyte
proliferation and migration.
Presence of the LEE (locus of enterocyte
effacement) in pig attaching and effacing Escherichia coli and characterization of eae, espA, espB and espD genes of PEPEC (pig EPEC) strain 1390.
Zinc oxide protects cultured enterocytes
from the damage induced by Escherichia coli.
Recently, we have reported that weaning may induce enterocyte
apoptosis through the activation of Fas-dependent and mitochondria-dependent apoptosis .
is informed of body requirements for iron and programmed accordingly at the base of the crypt.
Reports have also shown that lectins bind to complementary carbohydrates present on the surface of enterocytes
, for example in glycolipids and glycoproteins of the brush border membrane and as a result of enterocyte
atrophy, the nutrient absorption surface area becomes reduced and the transport of nutrients through the epithelium gets impeded, which leads to inhibited growth of animals.
TLR-4 mediates phagocytosis and translocation of bacteria across the intestinal barrier.
Group D animals had significantly greater increase in enterocyte
proliferation (indicating healing from damage) than animals in Group C.
The normally encoded protein regulates enterocyte
export in response to hepcidin, while the mutant is resistant to internalisation and degradation, resulting in unregulated iron absorption.
It can present many absorption characteristics analogous to intestinal cells during culture, such as formatting a monolayer of the cells and expressing several morphological and functional characteristics of the mature enterocyte
(Rousset, 1986; Artursson, 1990; Gan et al.
Subadult abalone did not demonstrate the same response in the crop epithelium as juvenile abalone, and this could be a result of the enterocyte
turnover rate, which could be masking the effects of epithelial hypertrophy in subadults compared with juveniles.
These glucosidases are comprised of two enterocyte
membrane bond complexes of N- and C-terminals each, as well as of maltase-glucoamylase (MGAM) and sucrase-isomaltase, making them the ultimate step in producing dietary glucose.