endocytosis


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endocytosis

 [en″do-si-to´sis]
the uptake by a cell of material from the environment by invagination of the plasma membrane; it includes both phagocytosis and pinocytosis.
Endocytosis. Shown are pinocytosis of small fluid droplets (left) and phagocytosis of a large particle (right). From Dorland's, 2000.

en·do·cy·to·sis

(en'dō-sī-tō'sis),
Internalization of substances from the extracellular environment through the formation of vesicles formed from the plasma membrane. There are two forms: (a) fluid phase (pinocytosis), and (b) receptor mediated.
See also: phagocytosis. Compare: exocytosis (2).
[endo- + G. kytos, cell, + -osis, condition]

endocytosis

(ĕn′dō-sī-tō′sĭs)
n.
A process of cellular ingestion by which the plasma membrane folds inward to bring substances into the cell.

en′do·cyt′ic (-sĭt′ĭk), en′do·cy·tot′ic (-sī-tŏt′ĭk) adj.
en′do·cy·tose′ (-tōs′) v.

en·do·cy·to·sis

(en'dō-sī-tō'sis)
Internalization of substances from the extracellular environment through the formation of vesicles formed from the plasma membrane.
See also: phagocytosis
Compare: exocytosis (2)
[endo- + G. kytos, cell, + -osis, condition]

endocytosis

A method by which a large molecule, such as a protein, can enter a cell. The plasma membrane of the cell is invaginated by the molecule so that an internal vesicle is formed containing the molecule. This then breaks off from the surface membrane and moves into the interior of the cell. Such vesicles are coated with a protein called clathrin.

endocytosis

an active process by which some cells can enclose a smaller body (e.g. a food particle) forming a membrane-bound vesicle. Compare EXOCYTOSIS. See PHAGOCYTOSIS, PINOCYTOSIS.
References in periodicals archive ?
According to our PPI scores, the TIM type receptor was the highest for the ZIKV clathrin-independent endocytosis mechanisms.
Large proteins and algae fragments are likely to be taken up into ventral epithelial cells by endocytosis or phagocytosis, since these cells rapidly endocytose bath-applied ferritin (Ruthmann et al..
Endocytosis of G protein-coupled receptors: roles of G protein-coupled receptor kinases and beta-arrestin proteins.
Pathway#: 1 = cell extracellular matrix interactions; 2 = alpha-linolenic acid metabolism; 3 = metabolism of lipids and lipoproteins; 4 = VEGF signaling pathway; 5=P75NTR recruits signaling complexes; 6 = endocytosis; 7 = P75NTR signals via NFKB; 8 = hormone ligand binding receptors; 9 = set pathway; 10 = linoleic acid metabolism; 11 = notch HLH transcription pathway; 12 = long term depression; 13 = FC epsilon RI signaling pathway; 14 = AKAP centrosome pathway; 15 = NA CL dependent neurotransmitter transporters; 16 = activated TAK1 mediates P38 MAPK activation; 17 = ABC transporters; 18 = amino acid transport across the plasma membrane; 19 = amyotrophic lateral sclerosis (ALS); 20 = other glycan degradation.
Aluminium uptake takes place by transferrin-receptor-mediated endocytosis and of aluminum citrate by system Xc and an organic anion transporter [132].
The actin cytoskeleton functions in the generation and maintenance of cell morphology and polarity, in endocytosis and intracellular trafficking, contractility, motility, and cell division.
Localization of AgNPs inside the cells may answer this issue since PBMCs are notorious for their insufficient cellular systems prepared for an endocytosis. Internalized particles by a membrane rupture would be strong evidence that the PBMCs are lysed by Ag particles.
However, whether ATPase-B1 interacts with them in HDL endocytosis is still unclear.
These findings suggested that, even with MSN-PdTPP's appreciable generation of [sup.1][O.sub.2], PDT therapeutic benefit strongly depended upon efficient nanoplatform endocytosis.
We used immunocytochemical assessment of NK-1R endocytosis in myenteric plexus neuronal cell bodies as an index of endogenous SP release and as an index of the action of locally released SP as described previously [14, 17, 20, 23].
The tumor cell then folds its membrane in to enclose the receptor and prodrug in a bubble and bring them into the cell (receptor-mediated endocytosis).
The uptake of large amounts of lipids from plasma lipoproteins via endocytosis by macrophages in the artery wall to form lipid-engorged foam cells is an important initiating event in the development of atherosclerosis, a major cause of heart disease.