elastase


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elastase

 [e-las´tās]
an enzyme capable of catalyzing the digestion of elastic tissue.

e·las·tase

(ē-las'tās),
A serine proteinase hydrolyzing elastin; other elastase-like enzymes have been identified, for example, pancreatic elastase (pancreatopeptidase E) and leukocyte elastase (lysosomal or neutrophil elastase), with different sequences and kinetic parameters; all have fairly broad specificities.

elastase

(ĭ-lăs′tās, -tāz)
n.
Any of a class of enzymes that catalyze the hydrolysis of elastin, especially one that is found in pancreatic juice.

elastase

(1) Pancreatic elastase, EC 3.4.21.11. 
(2) Leukocyte elastase, EC 3.4.21.37.

e·las·tase

(ĕ-las'tās)
A serine proteinase hydrolyzing elastin.
References in periodicals archive ?
aeruginosa from human immune response.23 The elastase enzyme damages tissues and degrades plasma proteins such as complement factors and immunoglobulins which are vital for the immune system of host.24 A high frequency of plcH, plcN and exoS was observed in the current study.
Effect of sub-MIC dose (500 pg/ml) of P tenuiflorus extract on production of QS regulated virulence factors such as pyocyanin, Staphylolytic activity, LasB elastase, LasA protease and chitinase activity Pyocyanin 71.96 Staphylolytic 18.21 Las B Elastase 38.74 Las A Protease 30.84 Chitinase 44.75 Note: Table made from bar graph.
Neutrophil elastase in cyclic and severe congenital neutropenia.
Suicidal NETosis is regulated by NADPH, myeloperoxidase, and elastase and is induced by microorganisms and chemical agents such as PMA [1,6,7].
In addition, our patients were not tested for antibodies to other p-ANCA antigens such as elastase and lactoferrin.
Changes in plasma biomarkers related to lung tidal volume changes, including plasma neutrophil elastase and CC16, are shown in Figure 3.
Stool samples for fecal elastase 1 determination, with firm consistency (not loose or watery), were collected at home or in the hospital.
We report that intratracheal administration of lung-derived MSCs ameliorated alveolar damage induced by elastase. This effect may have been mediated by the release of HGF as MSC-dependent paracrine mechanisms.
AAT augmentation only helps to supply SerpinA1 to the lungs where it can help prevent neutrophil elastase damage.