eNOS


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eNOS

(ē'nos),
Abbreviation for endothelial nitric oxide synthase.

NOS3

A gene on chromosome 7q36 that encodes endothelial nitric oxide synthase (eNOS), a small molecule implicated in vascular smooth muscle relaxation through a cGMP-mediated signal transduction pathway. eNOS is a critical mediator of cardiovascular homeostasis that regulates the diameter of blood vessels and keeps vascular proliferation and apoptosis in check. It is upregulated by dynamin, porin and soluble guanylyl cyclase, and downregulated by interactions with Hsp90 and Cav-1 in the Golgi and by NOSIP and NOSTRIN at the plasma membrane. NOS3 is also a less preferred gene symbol for what is now designated as NANOS3, see there.
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Levels of NO, eNOS, MDA, SOD3, and Left Ventricular Ejection Fraction of the Patients.
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The mRNA expressions of COX-2, PGIS, iNOS, eNOS, HIF-1a, HIF-2a, and VEGF were quantified by real-time RT-PCR.
Tongxinluo Inhibits Cyclooxygenase-2, Inducible Nitric Oxide Synthase, Hypoxia-inducible Factor-2a/Vascular Endothelial Growth Factor to Antagonize Injury in Hypoxia-stimulated Cardiac Microvascular Endothelial Cells
However, the major physiological source of NO in a normal, adult, and unstrained cardiac tissue is most likely eNOS originating from EE and MVE cells, while NO originating from cardiomyocytes is probably negligible if they are not activated.
Modulatory role of nitric oxide in cardiac performance/Modulatorna uloga azot-oksida na srcane performance
The aim of the present study was to investigate the possible association between eNOS 4a/b genotypes and alleles with the risk of preeclampsia.
Endothelial nitric oxide synthase (eNOS) 4a/b and G894T polymorphisms and susceptibility to preeclampsia
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PJ elicited no effects on eNOS protein expression or catalytic activity.
Pomegranate juice protects nitric oxide against oxidative destruction and enhances the biological actions of nitric oxide
ON THe TRaIL: Joel Kinnaman (Detective Stephen Holder) and Mireille enos
Murder most foul
The eNOS gene, as a candidate gene in the investigations on hypertension genetics, has attracted the attention of several researchers because of the established role of NO in vascular homeostasis.
Endothelial nitric oxide synthase (eNOS) variants in cardiovascular disease: pharmacogenomic implications

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