dysmorphogenesis

dys·mor·pho·gen·e·sis

(dis'mōr-fō-jen'ĕ-sis),
The process of abnormal tissue formation.
[dys- + G. morphē, form, + genesis, production]
Farlex Partner Medical Dictionary © Farlex 2012

dys·mor·pho·gen·e·sis

(dis'mōr-fō-jen'ĕ-sis)
The process of abnormal tissue formation.
[dys- + G. morphē, form, + genesis, production]
Medical Dictionary for the Health Professions and Nursing © Farlex 2012

dysmorphogenesis

Severe abnormality of body development caused by influences operating at an early stage of fetal growth. Monstrous maldevelopment (TERATOGENESIS).
Collins Dictionary of Medicine © Robert M. Youngson 2004, 2005
References in periodicals archive ?
The precise pathogenesis of NCM remains incompletely understood, but it is thought to be related to a congenital dysmorphogenesis of neural crest-derived pluripotent precursor cells that normally migrate from the neuroectoderm to the meninges and skin.
Free radical scavenging enzymes in fetal dysmorphogenesis among offspring of diabetic rats.
These anomalies in the disorganization of the ventricular myocardium resembled those seen in the human cardiomyopathy - noncompaction of ventricular myocardium.[24] In comparison with the treated group, cardiac abnormalities were sparse in control groups, only two cases of atrial septal dysmorphogenesis being observed [Table 1].{Table 1}{Figure 1}
The increase in cPKC[gamma] activity is also associated with diabetic embryonic dysmorphogenesis in rat [22].
[1-3] In general, the FA phenotype is characterised by a broad spectrum of congenital anomalies, principally involving growth, skin pigmentation and dysmorphogenesis of the skeletal, cardiovascular, genito-urinary, gastrointestinal and central nervous systems (CNS).
JNK's role in the regulation of glutamatergic synapse dysfunction and dysmorphogenesis is intriguing and not completely established.
Eriksson, "Receptor for advanced glycation end products (RAGE) knockout reduces fetal dysmorphogenesis in murine diabetic pregnancy," Reproductive Toxicology, vol.
(150) With respect to peroxisomal dysmorphogenesis in humans, only three patients have been identified with a different defect in two proteins involved in the proliferation and division of peroxisomes.
In utero and lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces genital dysmorphogenesis in the female rat.
Possibly, in analogy with schizophrenia, early cerebrocraniofacial dysmorphogenesis reflects an early stage of pathogenesis that occurs before behavioral manifestations [41].