Your doctor will want to monitor you for any breathing problems during your treatment with BOTOX for
detrusor overactivity associated with a neurologic condition.
Examples of such pre-operative comorbidities include pre-existing voiding dysfunction such as ISD (18), (19) or neurogenic
detrusor overactivity, as in Parkinson's disease or spinal cord injury.
Amundsen and her colleagues had expected to find notably lower nighttime urinary volumes in the participants with
detrusor overactivity because of a greater voiding frequency.
Women were excluded if they had
detrusor overactivity or previous sling surgery.
Additionally, oxybutynin is indicated in the treatment of pediatric patients aged 6 years and older with symptoms of
detrusor overactivity associated with neurological conditions (i.
org) recruited patients with refractory urge urinary incontinence and urodynamic evidence of
detrusor overactivity incontinence (DOI) to a 2:1 placebo controlled cystoscopic injection of botulinum toxin A.
It appears, however, that detrusor contractility bladder capacity, and the ability to withhold voiding decline in both sexes; the maximal urethral closure pressure and length probably decline in women;
detrusor overactivity increases in prevalence; and the post voiding residual (PVR) urine volume probably increases to 50 mL to 100 mL.
Your doctor will want to monitor you for any breathing problems during your treatment with BOTOX[R] for upper limb spasticity or for
detrusor overactivity associated with a neurologic condition.
The factors analyzed to determine association with acute urinary retention or the inability to voluntarily pass urine included age, gender, diagnosis of diabetes, baseline post void residual presence of urodynamic
detrusor overactivity, bladder capacity, peak flow rate and BTX-A dose.
is pleased to announce that BOTOX has been licensed by the Medicines and Healthcare Products Regulatory Agency (MHRA) for the management of urinary incontinence in adult patients with neurogenic
detrusor overactivity (NDO) due to subcervical spinal cord injury (SCI)(traumatic or non-traumatic) or multiple sclerosis (MS), who are not adequately managed with anticholinergics[sup.
6 shows marked neurogenic
detrusor overactivity with reduced compliance and attendant urine incontinence with each wave of instability.
Significant decrease in maximum detrusor pressure and increase in bladder compliance had been demonstrated in SCI patients with
detrusor overactivity by furosemide-stimulated filling cystometry (FCMG), which is a more physiologic method when compared with continuous CMG with a filling rate of 50 ml/min (1).