delayed conduction

de·layed con·duc·tion

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In the study conducted by Tandon et al., P100 latency was prolonged in hypertensives and suggested that hypertensive milieu affects neuronal excitation/conduction in the visual pathway.[2] In the study conducted by Marsh et al., P100 latency of visual evoked response was delayed in pre-eclamptic women.[8] There was a delayed conduction in brainstem auditory evoked response in Grade III hypertension.[9] There was impairment in sensory conduction in human beings in hypertension.[10] P100 latency was prolonged in patients with demyelinating disease like multiple sclerosis.[11] There is a significant change in VEP with normal fluctuation in carotid pressure and heart rate.[12] These findings were similar to the results of the present study.
Left ventricular hypertrophy results in myocardial fibrosis which through gap junctions and ion channel remodelling provokes significant electrophysiological changes which lead to delayed conduction velocity.
Increased arterial pressure in hypertension results in myocardial fibrosis, a high resistivity area having delayed conduction velocity and prolonged action potential duration.
Such heterogeneity can produce areas of unidirectional block and delayed conduction and lead to re-entry and TdP initiated by repetitive PVCs induced by triggered activity.
Hypokalaemia (<3 mmol/l) increases the cell membrane potential and is associated with delayed conduction causing atrial and ventricular ectopy, atrial tachycardia and AV block.
Kasai et al., (2001) in patients with supraventricular arrhythmias, documented evidence of delayed conduction and a propensity to conduction block within the CS.
Katritsis et al., (2002) also found delayed conduction in the CS and associated these findings with an increased proclivity to atrial arrhythmias.
At the next node, depolarization occurs again, facilitated by the presence of sodium channels, to propagate the impulse further.[28] Thus, a disruption in the myelin sheath causes delayed conduction velocity, or conduction block.
observed a higher threshold for evoking the V wave of BAEP and a higher absolute peak latencies of waves with a decrease in amplitude of wave V during premenstrual phase and have attributed this to the delayed conduction time of auditory impulses from auditory nerve to midbrain.