deaminase


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deaminase

 [de-am´ĭ-nās]
an enzyme causing deamination (removal of the amine group from a compound); enzymes are named according to substrate, such as adenosine deaminase, cytidine deaminase, guanine deaminase, etc.

de·am·i·nase

(dē-am'ĭ-nās),
Any enzyme that removes an amino group from a compound.
[de- + amino + -ase]

deaminase

/de·am·i·nase/ (de-am´ĭ-nās) an enzyme causing deamination, or removal of the amino group from organic compounds, usually cyclic amidines.

deaminase

(dē-ăm′ə-nās′, -nāz′)
n.
Any of a class of enzymes that catalyze the removal, usually by hydrolysis, of the amino group NH2 from an amino compound.

deaminase

[dē·am′inās]
Etymology: L, de, away, amine, ammonia; Fr, diastase, enzyme
one of the subclasses of enzymes that catalyze the hydrolysis of the NH2 bond in amino compounds, usually with the concomitant removal of ammonia. The enzymes are usually named according to the substrate, such as adenosine deaminase, guanine deaminase, or guanosine deaminase. Also called aminohydrolase.

deaminase

An ENZYME that brings about the breakdown of amino compounds.

deaminase

an enzyme causing deamination, or removal of an amino group from organic compounds, named according to its substrate as adenosine deaminase, cytidine deaminase, guanine deaminase, etc.
References in periodicals archive ?
Adenosine deaminase in the diagnosis of tuberculous pleural effusion.
Combined use of pleural adenosine deaminase with lymphocyte/neutrophil ratio: increased specificity for the diagnosis of tuberculous pleuritis.
Researchers have recently identified cytokinin, gibberellin, auxin and ACC deaminase accumulation by PGPR (Timmusk and Wagner, 1999).
Lymphocytic pleural effusion and change of the adenosine deaminase cut-off level in tuberculosis diagnosis.
Allosteric inhibition of human lymphoblast and purified porphobilinogen deaminase by protoporphyrinogen and coproporphyrinogen.
Since 1978, adenosine deaminase (ADA) has been used as a diagnostic test in tuberculous effusions.
GlaxoSmithKline (GSK), Fondazione Telethon (Telethon) and Ospedale San Raffaele (OSR) today announced that the European Commission has approved Strimvelis, the first ex-vivo stem cell gene therapy to treat patients with a very rare disease called ADA-SCID (Severe Combined Immunodeficiency due to Adenosine Deaminase deficiency).
A Hospital Based Study on Estimation of Adenosine Deaminase Activity in Cerebrospinal Fluid in Various Types of Meningitis.
Adenosine deaminase in experimental trypanosomiasis: future implications.