pertaining to or connecting the cerebral cortex and the medulla oblongata or brainstem.
Miller-Keane Encyclopedia and Dictionary of Medicine, Nursing, and Allied Health, Seventh Edition. © 2003 by Saunders, an imprint of Elsevier, Inc. All rights reserved.

cor·ti·co·bul·bar fi·bers

term formerly used to describe projections of the motor and sensory cortices to nuclei of the rhombencephalon innervating the musculature of the face, tongue, and jaws and some fibers to rhombencephalic relay nuclei; replaced by bulbar corticonuclear fibers (to medulla), pontine corticonuclear fibers (to pons), and mesencephalic corticonuclear fibers (to midbrain). See these individual entries.
Farlex Partner Medical Dictionary © Farlex 2012


Pertaining to corticofugal fibers projecting to the rhombencephalon that terminate 1) directly on some motor cranial nerve nuclei, 2) in the reticular formation, or 3) on sensory relay nuclei, such as the cuneate, gracile, and spinal trigeminal nucleus.
Medical Dictionary for the Health Professions and Nursing © Farlex 2012
References in periodicals archive ?
bulbar palsy, corticobulbar tract, or myelography (Federative International Programme on Anatomical Terminologies, 2017).
Harris et al., "Characterization of corticobulbar pharyngeal neurophysiology in dysphagic patients with Parkinson's disease," Clinical Gastroenterology and Hepatology, vol.
Hemiplegia is associated with a reduced number of functional corticospinal and corticobulbar fibers to the spinal and brainstem because of lesions in the cerebral cortex.
Brain damage causes spasticity due to the disruption of facilitatory corticobulbar fibers and causes inhibition of ventromedial reticular formation [3].
This was probably due to Wallerian degeneration and/or to arrested development of the corticospinal and corticobulbar tracts, as well as pontocerebellar connections, which were well demonstrated by neurofilament immunostaining (Figure 6, C and D).
PBA is thought to occur as a result of injury or disease that disrupts pathways regulating emotional expression, or affect, including the corticobulbar tracts and basal ganglia.
LIS was originally introduced by Plum and Posner in 1966 as a condition associated with lesion of the ventral pons, disrupting the corticospinal and corticobulbar pathways without involvement of the cortex [8].
Wilson (7) in 1924 proposed that pathological laughing and crying (PLC) was related to motor disinhibition resulting from bilateral corticobulbar lesions that disengaged a brainstem "faciorespiratory center" from cortical levels of control.
Its really interesting and exciting effect is the hypothesis that the pharyngeal electrical stimulation causes corticobulbar excitability change and cortical reorganization, resulting in an increase in neural plasticity (2,3,8,12).
(3) Larger lesions extend laterally while sparing the corticospinal and corticobulbar tracts.