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 [kor´teks] (pl. cor´tices) (L.)
the outer layer of an organ or other structure, as distinguished from its inner substance or medulla. adj., adj cor´tical.
adrenal cortex (cortex of adrenal gland) the outer, firm layer comprising the larger part of the adrenal gland; it secretes mineralocorticoids, androgens, and glucocorticoids.
cerebellar cortex the superficial gray matter of the cerebellum.
cerebral cortex (cortex cerebra´lis) the convoluted layer of gray matter covering each cerebral hemisphere. See also brain.
renal cortex the granular outer layer of the kidney, composed mainly of glomeruli and convoluted tubules, extending in columns between the pyramids that constitute the renal medulla.
striate cortex part of the occipital lobe that receives the fibers of the optic radiation and serves as the primary receiving area for vision. Called also first visual area.
visual cortex the area of the occipital lobe of the cerebral cortex concerned with vision; the striate cortex is also called the first visual area, and the adjacent second and third visual areas serve as its association areas.
Miller-Keane Encyclopedia and Dictionary of Medicine, Nursing, and Allied Health, Seventh Edition. © 2003 by Saunders, an imprint of Elsevier, Inc. All rights reserved.


Plural of cortex.
Farlex Partner Medical Dictionary © Farlex 2012


Plural of cortex.
Medical Dictionary for the Health Professions and Nursing © Farlex 2012
References in periodicals archive ?
The most evident differences were found in counts of cells marked with anti-GFAP in both sexes along the primary motor and hippocampus cortices. The hippocampus is recognized as one of the principal areas in control of the response to stress, learning, and memory [27].
For male subjects, there was an increase in GFAP-marked cells in primary motor cortices, M1 and M2; in contrast, for female subjects, there was a GFAP-marked cells count decrease in these same areas.
Functional Interplay between Primary Sensory Cortices
Although these electrophysiological studies provide evidence for a functional interplay between A1 and V1, it remained unclear, however, how this interplay is reflected at the systemic level of these cortices.
In summary, these studies highlight the ability of the juvenile and adult deprived cortex that neuronal activity from spared sensory modalities can "hitchhike" the deprived sensory cortices. Such changes are broadly referred to as "cross-modal recruitment" which represents one category of cross-modal plasticity [35].
Recent studies extended the view of cross-modal recruitment by demonstrating that the loss of one sense also provokes massive plastic changes in the spared sensory cortices. For instance, it has been shown that only one week of visual deprivation in juvenile mice acts to sharpen the tuning of layer 2/3 cells in the barrel field of S1 [58].
As they got bigger, their sensory and motor cortices barely expanded.
Our association cortices are crucial for the kinds of thought that we humans excel at.
Association cortices are also unusual for their wiring.
In the human brain, some neurons still receive chemical signals that cause them to form a bucket brigade from the sensory cortices to the motor cortices.