copper deficiency


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copper deficiency

The clinical consequences of inadequate consumption or absorption of dietary copper. Its hallmarks include an unsteady gait, neuropathy, muscle spasticity, and, occasionally, anemia. It may occur as a consequence of gastric bypass surgery or long-term parenteral nutrition.
See also: deficiency

copper deficiency

This rare condition occasionally affects young children, causing retardation of growth, rarefaction of bones and anaemia.
References in periodicals archive ?
This report described other peripheral neurologic condition related to the copper deficiency that was characterized mainly by monoparesis due to a neurogenic atrophy of an extensor muscle.
1992) showed that copper deficiency causes hypercholesteromy because of the increase in hepatic GSH with an increase in HMG-CoA reductase activity, which is the main enzyme that regulates the synthesis of cholesterol.
Over the last decade, the relationship between acquired copper deficiency and neurologic disease has become firmly established.
Secondary copper deficiency, chromium deficiency and trace element imbalance in the moose (Alces alces L.
The influence of experimentally induced copper deficiency on the fertility of rams.
Copper deficiency could be a result of either inadequate dietary intake (also termed primary copper deficiency) or due to impairment in its uptake (secondary copper deficiency).
Associations between copper deficiency and impaired brain function were noted nearly 75 years ago.
Evidence suggests that copper deficiency may be associated with heart disease, osteoarthritis, osteoporosis, microcytic hemolytic anemia, neutropenia, hypothermia, inappropriate immune system function, and bone demineralization.
Copper deficiency can have a dramatic effect in the young growing animal, the effects in adults though are often more insidious but have major effects on productivity.
Copper (in the form of cupric acid) was added to the AREDS formulations containing zinc to prevent copper deficiency anemia, a condition associated with high levels of zinc intake.
A copper deficiency may produce symptoms of retarded growth, impaired pigmentation, anemia, impaired reproductive function, fragile bones, and impaired immune function.
Researchers claim this finding supports the hypothesis of a mild copper deficiency in most Alzheimer patients.