This report described other peripheral neurologic condition related to the
copper deficiency that was characterized mainly by monoparesis due to a neurogenic atrophy of an extensor muscle.
1992) showed that
copper deficiency causes hypercholesteromy because of the increase in hepatic GSH with an increase in HMG-CoA reductase activity, which is the main enzyme that regulates the synthesis of cholesterol.
Over the last decade, the relationship between acquired
copper deficiency and neurologic disease has become firmly established.
Secondary
copper deficiency, chromium deficiency and trace element imbalance in the moose (Alces alces L.
The influence of experimentally induced
copper deficiency on the fertility of rams.
Copper deficiency could be a result of either inadequate dietary intake (also termed primary
copper deficiency) or due to impairment in its uptake (secondary
copper deficiency).
Associations between
copper deficiency and impaired brain function were noted nearly 75 years ago.
Evidence suggests that
copper deficiency may be associated with heart disease, osteoarthritis, osteoporosis, microcytic hemolytic anemia, neutropenia, hypothermia, inappropriate immune system function, and bone demineralization.
Copper deficiency can have a dramatic effect in the young growing animal, the effects in adults though are often more insidious but have major effects on productivity.
Copper (in the form of cupric acid) was added to the AREDS formulations containing zinc to prevent
copper deficiency anemia, a condition associated with high levels of zinc intake.
A
copper deficiency may produce symptoms of retarded growth, impaired pigmentation, anemia, impaired reproductive function, fragile bones, and impaired immune function.
Researchers claim this finding supports the hypothesis of a mild
copper deficiency in most Alzheimer patients.
