conduction block


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con·duc·tion block

failure of impulse transmission at some point along a nerve fiber, although conduction along the segments proximal and distal to it are unaffected; clinically, most often the result of an area of focal demyelination or, less often, transient ischemia; when caused by focal trauma involving the periperal nervous system, called neurapraxia.

con·duc·tion block

(kŏn-dŭkshŭn blok)
Failure of impulse transmission at some point along a nerve fiber, although conduction along the segments proximal and distal to it are unaffected; clinically, most often the result of an area of focal demyelination or, less often, transient ischemia.
References in periodicals archive ?
Additionally, in n-hexane neuropathy, decreased motor nerve conduction velocity with focal conduction block followed by dramatically diminished compound muscle action potentials (CMAPs) has been shown [13].
The first examination revealed partial motor conduction block in the wrist-elbow segments of both median nerves and the knee-ankle segment of right tibial nerve (Figure 1a-b-c).
Necessity for biatrial ablation to achieve birectional cavotricuspid isthmus conduction block in a patient following Senning operation.
We showed that the IgG monoclonal anti-GD1a antibody injected into rat sciatic nerves caused deposition of IgG and complement products on the nodal axolemma and disrupted clusters of nodal and paranodal molecules predominantly in motor nerves, and induced early reversible nerve conduction block. (107) Injection of IgG monoclonal anti-GD1b antibody induced nodal disruption predominantly in sensory nerves.
Reversal of sinus arrest and atrioventricular conduction block in patients with Sleep Apnea during Nasal continuous positive pressure.
Proximal slowing in carpal tunnel syndrome resulting from either conduction block or retrograde degeneration.
FTY720-related adverse events included dose-related, transient, generally asymptomatic heart rate reduction, infrequent transient AV conduction block, mild (1-3 mm Hg) blood pressure increase, macular edema (more common with 1.25 mg than the 0.5 mg target dose), and asymptomatic, reversible elevation of liver enzymes.
Electromyography displayed signs of a peripheral neuropathy and evidence of a conduction block. At the time of hospital admission, antichikungunya IgM and IgG were detected in 2 serum samples.
Smith et al described the main mechanism of temperature depended conduction block in the central nervous system, whereby the "blocking temperature" depends on the degree of demyelination.