coagulopathy


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coagulopathy

 [ko-ag″u-lop´ah-the]
any disorder of blood coagulation.
consumption coagulopathy disseminated intravascular coagulation.
Miller-Keane Encyclopedia and Dictionary of Medicine, Nursing, and Allied Health, Seventh Edition. © 2003 by Saunders, an imprint of Elsevier, Inc. All rights reserved.

co·ag·u·lop·a·thy

(kō'ag-yū-lop'ă-thē),
A disease affecting the coagulability of the blood.
Farlex Partner Medical Dictionary © Farlex 2012

coagulopathy

Hematology A clotting defect in which bleeding does not stop in the usual time period Etiology Hemophilia, drug-induced defects–eg, aspirin, thrombocytopenia, liver disease, Von Willebrand's disease. See Consumption coagulopathy, DIC, Leukemic coagulopathy.
McGraw-Hill Concise Dictionary of Modern Medicine. © 2002 by The McGraw-Hill Companies, Inc.

co·ag·u·lop·a·thy

(kō-ag'yū-lop'ă-thē)
A disease affecting the coagulability of the blood.
Medical Dictionary for the Health Professions and Nursing © Farlex 2012

Coagulopathy

A disorder in which blood is either too slow or too quick to coagulate (clot).
Mentioned in: Cerebral Palsy
Gale Encyclopedia of Medicine. Copyright 2008 The Gale Group, Inc. All rights reserved.
References in periodicals archive ?
(1,2,6) Moreover, many cases of SHL are related to coagulopathy. Congenital abnormalities, such as hemophilia or vitamin K deficiency; poor general conditions, such as hypotension or sepsis and antiplatelet drugs like non-steroidal anti-inflammatory drugs can cause coagulopathy.
Coagulopathy in critically ill patients: part 1: platelet disorders.
There are several contraindications of ESWL treatment including pregnancy, an untreated urinary tract infection/urosepsis, decompensated coagulopathy, uncontrolled arrhythmia, and an abdominal aortic aneurysm >4.0 cm.[2]
The patient required large volumes of blood products for resuscitation despite lack of laboratory evidence of coagulopathy.
The mechanisms causing thrombocytopenia in viper envenomation include vasculitis, sequestration of platelets in inflammed tissue, and consumption of platelets with potentiated development of intravascular disseminated coagulopathy (DIC).
Health care providers should consider vitamin K-dependent coagulopathy in patients with unexplained bleeding and reported or suspected synthetic cannabinoid use.
The baseline characteristics collected for both groups included age, gender, weight, height, serum creatinine, and past medical history of stroke and/or coagulopathy. Information on oral anticoagulation was also collected, including medication, indication, dose, dosing regimen, and appropriateness assessed based on the patient's age, indication, and renal function.
Clinical condition of the patient should be looked into and in case of coagulopathy, one can go ahead and deliver the baby adopting an aggressive approach.
Fresh Frozen Plasma is frequently transfused to patients with mild to moderate elevations in Prothrombin Time (PT) under the twin assumptions that these tests imply a coagulopathy and that FFP transfusion will correct the coagulopathy.
We present herein a case of HLH in a premature infant presenting with disseminated intravascular coagulopathy (DIC) and liver failure.
As shown in Table 3 thrombocytopenia (platelets < 50 billion/L) or coagulopathy, active hemorrhage and end stage liver disease (INR > 1.5) were the frequently observed absolute contraindications that potentially prevent patients to receive thromboprophylaxis.
After the coagulopathy improved and the patient stabilized, lumbar puncture was performed with administration of intrathecal chemotherapy.