chronic glomerulonephritis


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chron·ic glo·mer·u·lo·ne·phri·tis

glomerulonephritis that presents with persisting proteinuria, chronic renal failure, and hypertension, of insidious onset or as a late sequel of acute glomerulonephritis; the kidneys are symmetrically contracted and granular, with scarring and loss of glomeruli and the presence of tubular atrophy and interstitial fibrosis.
Synonym(s): chronic nephritis

chronic glomerulonephritis

Chronic nephritis Nephrology An advanced kidney disease characterized by inflammation and slowly progressive renal failure Etiology Idiopathic–1/4, amyloidosis, diabetic nephropathy, focal segmental glomerulosclerosis, IgA nephropathy, lupus nephropathy, myeloma, rapidly progressive glomerulonephritis. See Diabetic nephropathy, Focal segmental glomerulosclerosis, IgA nephropathy, Lupus nephropathy, Rapidly progressive glomerulonephritis.
References in periodicals archive ?
In this study, more patients with chronic glomerulonephritis (50.2%) were seen than the ones with DN (0.7%).
We hypothesized that all those metabolic and functional disorders contribute to CKD progression more significantly in case of chronic glomerulonephritis as an initial form of the disease.
Group Tissue n Age Gender M (%) HBV-GN K 54 36.1 [+ or -] 12.7 35 (64.8) CGN K 25 38.2 [+ or -] 15.5 * 18 (72.0) ** Group AIM2 Positive rate (%) - + ++ +++ HBV-GN 10 26 18 00 81.4 CGN 24 1 0 4.0 *** HBV-GN, hepatitis B virus-associated glomerulonephritis; CGN: chronic glomerulonephritis; K: kidney; M: male; * compared with HBV-GN, (t = -1.909, P = 0.06); ** compared with HBV-GN, ([chi square] = 0.400, P = 0.527); *** compared with HBV-GN, ([chi square] = 38.746, P < 0.01).
CGN: chronic glomerulonephritis; HTN: hypertension; ADPKD: autosomal dominant polycystic disease; SLE: systemic lupus nephritis.
Splinter hemorrhages may also be associated with mitral stenosis, vasculitis, cirrhosis, trichinosis, scurvy, chronic glomerulonephritis, and Darier's disease.
Table 1 Primary Etiologies of Kidney Disease in Children Congenital Structural (59%) * Obstructive uropathy * Aplasia/hypoplasia/dysplasia * Reflux nephrology * Polycystic kidney disease * Prune belly syndrome Inherited (3%) * Familial nephritis * Cystinosis * Primary oxalosis Acquired Inflammatory/immunologic (14%) * Focal segmental glomerulosclerosis * Systemic lupus erythematosus * Renal infarct * Chronic glomerulonephritis Infectious (4%) * Pyelonephritis * Hemolytic uremic syndrome Note: Adapted from NAPRTCS (2003).
Underlying renal diseases included chronic glomerulonephritis in 102 patients (50.5%), hypertensive nephropathy in 23 (11.4%), analgesic nephropathy in 25 (12.4%), polycystic kidney disease in 14 (6.9%), obstructive uropathy in 9 (4.5%), and unknown diseases in 29 (14.4%).
3), urinary L-PGDS excretion was significantly increased in the patients with diabetic nephropathy (median, 632 mg/mol Cr; range, 98-3250 mg/ mol Cr; P <0.001), IgA nephropathy (median, 291 mg/ mol Cr; range, 21-1393 mg/mol Cr; P <0.05), membranous nephropathy (median, 321 mg/mol Cr; range, 40-1531 mg/mol Cr; P <0.01), focal segmental glomerulosclerosis (median, 250 mg/mol Cr; range, 2001988 mg/mol Cr; P <0.01), chronic glomerulonephritis (median, 389 mg/mol Cr; range, 40-616 mg/mol Cr; P <0.01), and chronic renal failure (median, 1008 mg/mol Cr; range, 204-3220 mg/mol Cr; P <0.001).
The recipient suffered from chronic glomerulonephritis, a progressive kidney inflammation usually resulting in permanent kidney failure.
For example, the sclerotic glomeruli in chronic glomerulonephritis become less cellular, more compact, and finally disappear.
Chronic glomerulonephritis was associated with microfilariasis in the brown capuchin monkey (Cebus apella) (Sanchez Negrette 1983) and this was the only report about this specie in spite of being utilized in biomedical and behavioral research (Nagle et al.
Groehler (1975), "Hydrocarbon Exposure and Chronic Glomerulonephritis," Lancet, 2(7927):199-201.

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