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invasion of the central nervous system by wandering nematode larvae; for example, Angiostrongylus cantonensis in rats and humans.
pertaining to the brain and spinal cord.
see brain abscess.
is thought to be a sequel to subclinical edema disease due to an Escherichia coli toxemia of pigs. The disease is sporadic within a group and is characterized by incoordination, apathy, aimless walking and circling. Emaciation occurs rapidly and most affected animals are euthanatized on humanitarian grounds.
a characteristic lesion in the brain of newborn piglets affected by congenital tremor caused by swine fever infection of the dam during early pregnancy. See also congenital tremor syndrome of piglets.
uncommon in animals. May result from marrow escaping into the circulation from a fracture site, or cartilage from a nucleus pulposus disruption. Usually in a ventral spinal artery causing a sudden onset of paralysis.
cerebrospinal fluid (CSF)
the fluid within the subarachnoid space, the central canal of the spinal cord, and the four ventricles of the brain. The fluid is formed continuously by the choroid plexus in the ventricles, and is reabsorbed into the blood by the arachnoid villi at approximately the same rate at which it is produced.
Examination of the CSF for the presence of abnormal or excessive numbers of cells, protein content, pressure is an important source of information about the nervous system.
cerebrospinal fluid-blood barrier
CSF passes into the CSF system (brain ventricles, the central canal of the spinal cord and the subarachnoid space) at the choroid plexus and passes out of the subarachnoid space into the sagittal sinus. The confining membranes of the system control selectively the passage of certain materials between it and the brain tissue (CSF-brain barrier) and between it and the blood (CSF-blood barrier).
cerebrospinal fluid-brain barrier
see cerebrospinal fluid-blood barrier (above).
invasion of the central nervous system by the microfilaria of Setaria labiatopapillosa (S. digitata) in most species causes an acute focal encephalomyelomalacia. The clinical picture is one of incoordination, then paralysis of the limbs, especially the hinds. S. equina may cause endophthalmitis in horses by similar invasion.
see cerebrospinal embolism (above).