cerebellar syndrome


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cer·e·bel·lar syn·drome

the signs and symptoms of cerebellar dysfunction: dysmetria, dysarthria, asynergia, nystagmus, ataxia, staggering gait, and adiadochokinesia.

cerebellar

pertaining to the cerebellum.

cerebellar abiotrophy
occurs in cattle, pigs and dogs. Affected young are normal at birth but at an early age ataxia and signs of cerebellar dysfunction appear, often progressing to complete immobilization. Cerebral function is usually normal. An inherited basis is suspected. In Kerry blue terriers, it is inherited as an autosomal recessive trait. Called also cerebellar neuronal abiotrophy.
cerebellar agenesis
absence of the cerebellum due to its non-appearance in the embryo.
cerebellar aplasia
see cerebellar atrophy (below).
cerebellar ataxia
the incoordination of gait characterized by exaggerated movements. There is no paresis. There is exaggerated strength and distance of movement—hypermetria. Caused usually by damage to the cerebellum or to the spinocerebellar tracts. May be congenital due to cerebellar atrophy or acquired due to inflammation or malacia of the cerebellum.
cerebellar atrophy
degeneration and loss of cells—Purkinje and granular cells of the cerebellum. Present at birth or soon after, is congenital in sheep, cattle, Arab horses, dogs and cats. Some of the diseases are inherited, some are known to be due to virus infection in utero, e.g. bovine virus diarrhea, feline panleukopenia. Some are in fact abiotrophies, premature aging of tissues. In the latter the animals are normal at birth but develop classical signs later. Segmental atrophy occurs in pigs but is asymptomatic.
cerebellar coning
see cerebellar lipping (below), brain herniation.
cerebellar cortex
the superficial gray matter of the cerebellum.
cerebellar dysfunction
see cerebellar ataxia (above).
cerebellar dysmelinogenesis
recorded in Chow Chow dogs; characterized by congenital head tremor.
feline cerebellar ataxia
see feline panleukopenia.
cerebellar hypomyelinogenesis
abnormally reduced myelination in the cerebellum; characterized clinically by severe neonatal tremor.
cerebellar hypoplasia
deficiency of cells of the cerebellum, the degree and distribution of which is variable. See cerebellar atrophy (above).
inherited cerebellar defects
includes cerebellar abiotrophy, atrophy, agenesis, hypoplasia, neuraxonal dystrophy.
cerebellar lipping
caused by diffuse cerebral edema. The vermis of the cerebellum protrudes through the foramen magnum and lies like a tongue over the medulla.
cerebellar neuronal abiotrophy
see cerebellar abiotrophy (above).
cerebellar neuraxonal dystrophy
reported in collie sheepdogs. The lesion is limited to axons and there are no lesions in the cerebellar folial neurons.
cerebellar syndrome
see cerebellar ataxia (above).
References in periodicals archive ?
The previous studies suggested that RBD was considered as a red flag for the diagnosis of MSA [sup][30] and might either antedate or follow the onset of parkinsonism, cerebellar syndrome, and dysautonomia [sup][31] that might be due to inchoate structural lesion which damages REM regulating regions of brain (lower brainstem and limbic system mainly).
Neurological examinations show cerebellar syndrome findings, oculomotor apraxia, slowing down in saccades, a reduction in vestibulo-ocular reflex, and reduction in optokinetic nystagmus.
Nonetheless, Gilman et al (6) lists specific criteria for probable MSA, including autonomic dysfunction, orthostatic hypotension, and either parkinsonism or cerebellar syndrome symptoms.
International Cooperative Ataxia Rating Scale for pharmacological assessment of the cerebellar syndrome.
Adult coeliac disease presenting as cerebellar syndrome.
8%) and cerebellar syndrome (69%), papilloedema was the commonest sigh, which was seen in 68.
Patients 1 and 4 had encephalitis with drowsiness, which for patient 4 was accompanied by cerebellar syndrome (dysarthria and dysmetria).
6-8) Patients with GCN exclusively presented cerebellar syndrome and cerebellar atrophy in MRI without white matter lesions.
Physical examination showed no pyramidal, vestibular, or cerebellar syndromes, and all tendon reflexes were typical.
Forty contributed chapters address anatomy and functions, imaging, malformations, genetic and metabolic disorders, cerebellar disruptions, vascular disorders, tumors, and paraneoplastci cerebellar syndromes, acute ataxia, and extrinsic insults.