cerebellar cortex


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cortex

 [kor´teks] (pl. cor´tices) (L.)
the outer layer of an organ or other structure, as distinguished from its inner substance or medulla. adj., adj cor´tical.
adrenal cortex (cortex of adrenal gland) the outer, firm layer comprising the larger part of the adrenal gland; it secretes mineralocorticoids, androgens, and glucocorticoids.
cerebellar cortex the superficial gray matter of the cerebellum.
cerebral cortex (cortex cerebra´lis) the convoluted layer of gray matter covering each cerebral hemisphere. See also brain.
renal cortex the granular outer layer of the kidney, composed mainly of glomeruli and convoluted tubules, extending in columns between the pyramids that constitute the renal medulla.
striate cortex part of the occipital lobe that receives the fibers of the optic radiation and serves as the primary receiving area for vision. Called also first visual area.
visual cortex the area of the occipital lobe of the cerebral cortex concerned with vision; the striate cortex is also called the first visual area, and the adjacent second and third visual areas serve as its association areas.

cer·e·bel·lar cor·tex

the thin gray surface layer of the cerebellum, consisting of an outer molecular layer or stratum moleculare [TA], a single layer of Purkinje cells (the Purkinje cell layer [TA] or stratum purkinjense [TA]), and an inner granular layer or stratum granulosum [TA].
Synonym(s): cortex cerebelli [TA]

cer·e·bel·lar cor·tex

(ser-ĕ-bel'ăr kōr'teks)
The thin gray surface layer of the cerebellum, consisting of an outer molecular layer or stratum moleculare, a single layer of Purkinje cells (the ganglionic layer), and an inner granular layer or stratum granulosum.
References in periodicals archive ?
We speculate that cTBS can have a similar effect when applied just before or just after EBCC, respectively, by decreasing the excitability of the cerebellar cortex, and thus hampering the formation of a new motor memory, or by interfering with the encoding of a newly formed memory pattern.
The analysis of the recorded electrocortical activity in the cerebral and cerebellar cortex was carried out in FORTRAN programs under DOS operating system and in the Matlab 6.5.
Arrows in (b) indicate the flow of signals in the cerebellar cortex. The CN neurons receive weak MF collaterals in the cerebrocerebellum and strong collaterals in other areas.
(6) In the cerebellum, during embryonic development, Purkinje cells, the principal neurons of and the only output neuron from the cerebellar cortex, migrate radially from the VZ towards the pial surface along the radial glia.
Previous studies have shown that the histamine-containing fibers project from the tuberomammillary nucleus to the cerebellar cortex and the deep cerebellar nucleus, with a high density of histaminergic terminations in the vermis and flocculus (6,7).
While we cannot rule out the possibility that some of the increased [alpha]3 immunolabeling in the Purkinje cell and molecular layers in the +/Lc cerebellar cortex may be in climbing fibers, it is clear that the intensity and distribution of [alpha]3 immunolabeling is altered in the +/Lc cerebellar cortex.
The cerebellar cortex, the brain region responsible for motor coordination and learning expresses a high density of GABARs.
The changes occurring in the number and diameter of the Purkinje cell of the cerebellar cortex with respect to age were studied.
The motor cortex, the cerebellar cortex controlling coordination and the visual cortex were dominant.
Changes in the microanatomy of the cerebellar cortex (Tables 5 and 6; Figure 5), the regression of germinal matrix in the paraventricular areas of the brain, the formulation of glomeruli in the kidneys, and the development of pulmonary acini are parameters that are useful in assessing developmental maturity.
Research by Dobbing and colleagues established the precept of a vulnerable period for neurodevelopment as reviewed elsewhere (Dobbing 1982), which in the human cerebellar cortex extends > 1 year after birth, rendering it susceptible to the effects of postnatal ETS (Dobbing 1982; Friede 1973; Koop et al.

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