Central venous oxygen saturations
. Value of serial determinations in patients with acute myocardial infarction.
APPENDIX 1 Glossary of terms CFI cardiac function index EDT exponential decay time EVLW extravascular lung water GEDV global end-diastolic volume GEF global ejection fraction ITBV intrathoracic blood volume MTT mean transit time PiCCO pulse index continuous cardiac output PVPI pulmonary vascular permeability index [S.sub.cv][O.sub.2] central venous oxygen saturation [S.sub.v][O.sub.2] mixed venous oxygen saturation
Resuscitation of the critically ill in the ED: responses of blood pressure, heart rate, shock index, central venous oxygen saturation, and lactate.
Table-III: Central venous oxygen saturation, lactic acid level and urine output before and after treatment (n=45, xs).
Central venous oxygen saturation, lactic acid level and urine output: Blood lactic acid level, central venous oxygen saturation index and urine output were also improved after treatment, with significant inter-group differences (Pless than 0.05) (Table-III).
Although central venous oxygen saturation has been shown to correlate well with mixed venous oxygen saturation during different phases of haemorrhagic shock, hypoxia and hyperoxia in an experimental model of circulatory failure (18), recent clinical studies showed that the agreement between the two oxygen saturation measurements can be very unpredictable in patients with septic and cardiogenic shock (19,20).
Fifth, this study did not assess whether using CVA-C[O.sub.2] in addition to central venous oxygen saturation will increase the ability of central venous oxygen saturation to predict the status of cardiac output in circulatory failure.
A normal central venous oxygen saturation (ScvO [sub]2 ) generally indicates that oxygen delivery (DO [sub]2 ) is sufficient to meet oxygen consumption (VO [sub]2 ), and further increasing DO [sub]2 is not necessary.
P(v−a)CO [sub]2 /C(a−v)O [sub]2 ratio is superior to central venous oxygen saturation