catecholaminergic

catecholaminergic

 [kat″ĕ-kol-am″in-er´jik]
activated by or secreting catecholamines.
References in periodicals archive ?
B65 was characterized previously as a bilaterally paired catecholaminergic neuron that projects an axon across the buccal commissure and exerts its strongest synaptic actions in the contralateral hemiganglion (Kabotyanski et al., 1998).
Gastric distension-induced c-fos expression in catecholaminergic neurons of rat dorsal vagal complex.
The effect of Clonidine on the hemodynamic responses may be attributable to its overall inhibitory action on the catecholaminergic areas of the lower brain, as stated by Aho M, et al (10) in 1990.
To the Editor: Catecholaminergic polymorphic ventricular tachycardia (CPVT) often occurs in children or adolescents without organic heart diseases.
While there is no study of NGF and GDNF in tic disorders, in a study investigated catecholaminergic genes there was a significant correlation between the BDNF gene expression and the severity of tic.18 In another study, there was no relationship between BDNF polymorphisms and tic severity.19 Similar to this study, no correlation was found between NGF and GDNF and tic severity, in our study.
Catecholaminergic polymorphic ventricular tachycardia is a rhythm disorder that develops in relation with genetic causes in the absence of structural heart disease.
Such relationship could have been due to the role of lactate as a signal for catecholaminergic activation as it has been observed for epinephrine in the crab Carcinus maenas (De Wacheter et al., 1997).
- Around 5 to 10 percent of sudden cardiac deaths are arrhythmias occurring in the absence of underlying structural heart disease (long QT syndrome, Brugada syndrome, Wolff-Parkinson-White syndrome, catecholaminergic polymorphic VT, all abnormalities seen on ECG).
Tomlinson, "Lewy body prevalence in the aging brain: relationship to neuropsychiatric disorders, Alzheimer-type pathology and catecholaminergic nuclei," Journal of the Neurological Sciences, vol.
Unlike acute exposure, long-standing catecholaminergic burden leads to structural myocardial alterations such as interstitial fibrosis, inflammation, and a subsequent dysfunction [10, 13].
Sympathetic neurons originate from trunk neural crest cells that arrest their migration upon arrival at the dorsal aorta and begin to express the catecholaminergic and noradrenergic biosynthetic enzymes tyrosine hydroxylase (TH) and dopamine betahydroxylase (DBH), respectively (Figures 1(a) and 1(b)).
Ware, "Paralogue annotation identifies novel pathogenic variants in patients with brugada syndrome and catecholaminergic polymorphic ventricular tachycardia," Journal of Medical Genetics, vol.