catabolite

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catabolite

 [kah-tab´o-līt]
a compound produced in catabolism.

ca·tab·o·lite

(kă-tab'ō-līt),
Any product of catabolism.

catabolite

(kə-tăb′ə-līt′)
n.
A substance produced by the process of catabolism.

ca·tab·o·lite

(kă-tab'ō-līt)
Any product of catabolism.
References in periodicals archive ?
The new equipments, as those for artificial respiration, cardiac resuscitation, for maintaining the function of the cord and lungs, of blood cleaning from toxic catabolites, are a few examples.
Iron deficiency induces a range of violations of free radical oxidation, as evidenced by the increase of catabolites of lipid peroxidation and the development of endogenous intoxication to raise the average molecular weight peptides.
Supplemental fructooligosaccharides and mannanoligosaccharides influence immune function, ileal and total tract nutrient digestibilities, microbial populations and concentrations of protein catabolites in the large bowel of dogs.
AHR agonists, including both endogenous (e.g., several tryptophan catabolites), dietary (e.g., indole-3-carbinol metabolite indolo[3,2b] carbazole), and xenobiotic [e.g., 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)] ligands, can dramatically impact host metabolism and immunity (Angrish et al.
Studies related with the levels of bone and collagen markers including bone specific ALP (B-ALP), type I collagen C terminal propeptide (PICP), type III procollagen, N terminal propeptide (P3NP), type I collagen terminal telopeptide (ICTP) and pyridolin (Pyd) and deoxypridolin which are catabolites of collagen are being conducted (6, 7).
Since all anabolites needed for foetal metabolism come from the mother s blood and foetal catabolites are passed back into the mother's circulation through the placenta, the examination of placenta gives a clear idea of what had happened with it, when it was in the mother's womb and what is going to happen with the foetus in future.
Under this situation, the organisms which were involved in the sedimentation process exhibit also terrigenous characteristics, and their dead bodies and catabolites have been deposited together with hydrothermal sediments according to [106].
If this occurs in MS patients too, a continuous outflow of ATP catabolites, including uric acid and its precursors, is expected from cerebral tissue of the MS lesions into the extracellular space.
In addition, cell-mediated immune cytokines can increase the synthesis of neurotoxic tryptophan catabolites (TRYCATs) which contributes to oxidative stress, impaired mitochondrial metabolism, and apoptosis.