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Condensation product of coenzyme A and n-butanoic acid; an intermediate in fatty acid degradation and in biosynthesis.
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References in periodicals archive ?
It is also likely that [H.sub.2]S disrupts fatty acid oxidation through inhibition of butyryl-CoA dehydrogenase.
Afterwards acetyl-CoAacetoacetyl-CoA and butyryl-CoA are formed from pyruvate as key intermediates in the main branch (Jones and Woods 1986).
Because urinary ethymalonic acid (EMA) elevation most likely reflects a cellular accumulation of butyryl-CoA (16), which is secondary to reduced SCAD catalytic activity, these patients are correctly considered as possibly having SCAD deficiency.
Ethylmalonic acid is observed due to defective butyryl-CoA dehydrogenase activity.