bradyphrenia

bradyphrenia

(brā-dē'frē-nē-a),
Slowness in mental processing due to a decreased ability to shift quickly from one conceptual pattern to another; most often seen with Parkinson disease.
[brady- + -phrenia]

bradyphrenia

(brăd″ē-frēn′ē-ă) [″ + Gr. phren, mind]
Slowness of thought and information processing, seen in some forms of dementia.
References in periodicals archive ?
3] Some of the associated features are bradymemia (slowness of thoughts), bradyphrenia and gait abnormalities, such as festination (shuffling gait).
Secondary factors that may contribute to bradykinesia include muscle weakness [60], rigidity [72], rest and action tremor [73], and movement variability and bradyphrenia [47].
Arciniegas and Anderson mentioned that the decrease in suicide attempts in people with PD may be related to the effects of bradykinesia, akinesia, bradyphrenia, and apathy.
Severe movement abnormalities and psychomotor deficits can sometimes be observed in patients with HAD, most notably in the form of bradykinesia (slowed movement), hypomimia, action/ postural tremor and hand agility, as well as bradyphrenia (slowed information processing).
Bradykinesia and bradyphrenia revisited: Patterns of subclinical deficit in motor speed and cognitive functioning in head-injured patients with good recovery.
Although they usually do not pose a significant problem for patients because they do not hinder day-to-day activities and responsibilities, subtle impairments such as bradyphrenia (slowness of thinking) and difficulty with finding the right word can also develop in PD.
Anhedonia, avolition affective blunting, alogia, asocialization, and bradyphrenia were negative symptom clusters considered to be fundamental defects by early investigators.
Hypothesis: The bradyphrenia of Parkinsonism is a nosological entity.
The lack of a simple model against which to test for the presence or absence of bradyphrenia was a major problem.
Then come five chapters on cognitive impairments (non-dementia), such as bradyphrenia and visuospatial abnormalities; six chapters on dementia in PD, including epidemiology, neuro-imaging and pathological correlations; four chapters on depression, including phenomenology, biochemistry and treatment, including, their effects on memory, affect and adverse neuropsychiatric complications.
49] Patients with IP who are not demented may have impairment of visuospatial function, executive function and memory, language disorders and bradyphrenia (slowing of thought processes).
Is the |lethargy of mind', bradyphrenia, accounted for by depression or dementia?