blind loop syndrome


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blind loop syn·drome

stagnation of intestinal contents with bacterial overgrowth, producing substances that interfere with absorption of fat, vitamins, and other nutrients; usually occurs in a portion of small intestine that has been excluded from the flow of chyme.
A complication of Billroth II subtotal gastroenterostomy—end-to-side enteroenteric anastomosis—that may arise long after surgery; the afferent loop consists of duodenum and a variable portion of jejunum, a loop that is a temporary reservoir for 1–1.5 litres of biliary and pancreatic secretions; after a fatty meal, the contents of a partially obstructed afferent loop increase and ‘explosively’ enter the stomach and may be regurgitated as greenish bilious fluid; other symptoms include intermittent diarrhoea due to disaccharidase deficiency, abdominal ‘colic’, haemorrhage, vitamin deficiency and neurologic symptoms; with prolonged partial obstruction, the stool is steatorrhoeic—bulky, grey, and greasy—accompanied by weight loss; complete blind loop obstruction may be a medical emergency with rapid deterioration, shock, and perforation peritonitis
Management Antibiotics—e.g., T-S—loop shortening, afferent-to-efferent or Roux-en-Y anastomoses or gastrojejunostomy

blind loop syndrome

Afferent loop syndrome, stagnant loop syndrome Surgery A complication of Billroth II subtotal gastroenterostomy–end-to-side enteroenteric anastomosis that may arise long after surgery; the afferent loop consists of duodenum and a variable portion of jejunum, a loop that is a temporary reservoir for 1-1.5 liters of biliary and pancreatic secretions; after a fatty meal, the contents of a partially obstructed afferent loop ↑ and 'explosively' enter the stomach and may be regurgitated as greenish bilious fluid; other Sx include intermittent diarrhea due to disaccharidase deficiency, abdominal 'colic', hemorrhage, vitamin deficiency and neurologic Sx; with prolonged partial obstruction, the stool is steatorrheic–bulky, gray, and greasy, accompanied by weight loss; complete blind loop obstruction may be a medical emergency with rapid deterioration, shock, and perforation peritonitis Treatment Antibiotics–eg, T-S, loop shortening, afferent-to-efferent or Roux-en-Y anastomoses or gastrojejunostomy

blind loop syn·drome

(blīnd lūp sin'drōm)
A group of symptoms that result from the overgrowth of bacteria (primarily anaerobic) in a surgically bypassed or disconnected segment ofintestine: local or systemic infection, fat malabsorption, and vitamin B12 and folate deficiencies.

blind loop syndrome

An uncommon disorder caused by stagnation of bowel contents as a result of adhesions, constrictions, pouches or other similar abnormalities. It features diarrhoea, fatty stools, abdominal pain, loss of weight, anaemia and vitamin deficiency.
References in periodicals archive ?
However, this procedure can be aggravating and become impossible due to adhesion formation.6 It also has a failure rate of 25%.2 Gastrojejunostomy, another procedure commonly employed provides adequate gastric decompression but can lead to incomplete release of duodenal obstruction leading to blind loop syndrome, gastric bile reflux and ulceration.6 Subtotal gastrectomy and Billroth II gastrojejunostomy, and repositioning of duodenum anteriorly are some of the otherless commonly used options when going for a surgical intervention.5 The aforementioned interventions proved successful in a study of seven patients by M.
A side-to-side anastomosis for proximal duplicate lumens was done to promote drainage and prevent blind loop syndrome. Appendicectomy was also done.
In upto 30% of patients symptoms may develop including chronic abdominal pain, malabsorption, blind loop syndrome, enterolith formation, haemorrhage, diverticulitis, obstruction, abscess formation and rarely diverticular perforation1,2,5,7,8.
Ischemic jejunal stenosis and blind loop syndrome after blunt abdominal trauma.
A predisposition to SIBO exists in diverse conditions where there is altered anatomy from prior surgery (eg, blind loop syndrome) or stricture or where there is impaired gut motility and prolonged orocecal transit time.
Pseudo-obstruction, blind loop syndrome, jejunal dyskinesia, chronic diverticulitis complicated by the formation of enterolith, and vitamin B-12 malabsorption secondary to chronic stasis and bacterial overgrowth within the jejunal diverticula have also been reported.