beta receptor

Also found in: Dictionary, Thesaurus.
Related to beta receptor: alpha receptor

beta receptor

A trio of G protein-coupled cell membrane receptors in the sympathetic nervous system, which dampen the response to catecholamines. They are divided into Beta-1, -2 and -3 receptors.

Beta-1 receptor increases cardiac output by raising the heart rate, impulse conduction, and contraction, thereby increasing the left ventricular ejection fraction; increases juxtaglomerular renin secretion; increases gastric secretion of ghrelin (the hunger hormone, which contrasts to leptin, the satiation hormone).

Beta-2 receptor prompts smooth muscle relaxation resulting in bronchodilation; reduced GI motility; relaxation of detrusor muscle of the bladder; glycogenolysis and gluconeogenesis; and increased renin secretion.

Beta-3 receptor increases lipolysis in fat and thermogenesis in skeletal muscle.
Segen's Medical Dictionary. © 2012 Farlex, Inc. All rights reserved.
References in periodicals archive ?
Pfeffer, "The lymphotoxin beta receptor controls organogenesis and affinity maturation in peripheral lymphoid tissues," Immunity, vol.
Corticosteroids can reverse the desensitization of beta receptors and are said to be able to potentiate the response to beta agonists.
Though mice without the beta receptor can live, they are plagued by a list of health problems--a list that continues to grow as researchers observe the mice aging.
Park et al., "Association between lymphotoxin beta receptor gene polymorphisms and IgA nephropathy in Korean children," Immunological Investigations, vol.
In fact function of alpha and beta receptors of estrogen influence on kidney cells.
There are three recognized subtypes of adrenergic beta receptors ([[beta].sub.1], [[beta].sub.2], [[beta].sub.3]) that can mediate the enhancement of insulin and glucagon secretion when activated by adrenalin, noradrenalin or adrenomimetics (NARIMIYA et al., 1981; MARCAL et al., 2006; AHREN, 2000).
Alpha receptors are the ones that cause relaxation of smooth muscles around bronchial tubes when stimulated, while stimulation of beta receptors increases heart activity.
The blood samples of the family members were sent to Chicago University, USA to search for possible genetic mutations in thyroid hormone beta receptors, since the laboratory findings in family members was compatible with PTHR
Wright explains that estrogen stimulation of alpha receptors tends to increase cell proliferation, while beta receptors tend to decrease it.
The team speculated that myocardial injury may be due to excessive stimulation of beta receptors, perhaps in combination with genetic predisposition to myocardial injury associated with that mechanism.
The fat level of our body is controlled by sets of cells called alpha and beta receptors.
Data from displacement binding assays using recombinant estrogen alpha and beta receptors will be presented.