autophagic


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Related to autophagic: autophagic vacuoles

au·to·pha·gic

(aw'tō-fā'jik),
Relating to or characterized by autophagia.

au·to·pha·gic

(aw'tō-fā'jik)
Relating to or characterized by autophagia.

autophagia

, autophagy (ot″ŏ-fā′j(ē-)ă) (o-tof′ă-jē) [ auto- + -phagy]
1. Biting oneself.
2. Self-consumption by a cell.
autophagic (ot″ŏ-fā′jik), adjective
References in periodicals archive ?
Autophagic activity is influenced by multiple factors including nutritional environment and amount of exercise, at least in humans and rodents [30,31].
Aside from the single bout exercise, long-term chronic exercise has also been shown to result in different autophagic response in skeletal muscle (Tam and Siu, 2014).
(16) In the neonatal rat ovary, autophagic cell death has been reported as an alternative mechanism for cell death in primordial follicle atresia.
Helicobacter pylori VacA induces autophagic cell death in gastric epithelial cells via the endoplasmic reticulum stress pathway.
LC3 is a cytosolic protein that when conjugated with a phosphatidylethanolamine is recruited to the autophagosome membrane favoring the autophagic influx (34).
Even though the process of autophagy may improve cell survival under harsh conditions, in many cases, autophagy can induce autophagic programmed cell death, which is also termed as type II cell death (15,16).
Although atorvastatin has been reported to have some autophagic effects on different kinds of cancer cell lines, there is no evidence in breast cancer (10,14).
Participants with type 1 diabetes had an increase in the size and number of autophagic remnants in muscles; however, levels of the autophagic regulatory protein, phosphorylated AMP-activated protein kinase, and its downstream targets were similar between groups.
Now, Sederma is reporting on its ability to protect and restore the autophagic activity of skin cells.
Thus, as previously we reported in one of our patients with Naxos disease, the enhanced automaticity of ectopic foci and/or reentry circuit around the scar tissue was abolished by cytoskeletal disruption mechanism, induced by tubulin destabilization with low-dose colchicines.6 Additionally interesting, colchicine also upregulates other proteins involved in the autophagic clearance of misfolded proteins such as the master regulator Transcription Factor EB (TFEB).
They cover enteroviruses future, Enterovirus receptors and entry, hijacking host functions for translation and RNA replication by enteroviruses, the omics of rhinoviruses, viral population dynamics and sequence space, Enterovirus control of cytoplasmic RNA granules, the autophagic pathway and Enterovirus infection, and the lipid blueprints of replicating viral genomes.