atrial conduction

atrial conduction

References in periodicals archive ?
found that the intraright atrial conduction time increased in patients with asthma.
No correlation was found between the duration of disease and [HbA.sub.1c], atrial conduction delay (r=0.003, p=0.493; r=-0.092, p=0.315, respectively).
In people, increases in duration and dispersion of P wave is associated with prolonged atrial conduction, left atrial remodeling and the predominant sympathetic tone, which impair the velocity of impulse propagation.
Prolonged atrial conduction times have been related to both onset and recurrence of atrial fibrillation [7, 8].
PQ prolongation shows increased atrial conduction time, which may be related to an increase in AF risk.
Tissue Doppler echocardiography is a beneficial diagnostic tool used to determine atrial conduction times.
A longstanding increase in blood pressure (BP) can lead to structural and electrical remodelling of the heart.[sup][1],[2],[3] It has been demonstrated that changes in atrial size and fibrosis cause dispersion of refractoriness and alterations in atrial conduction. As a result, arrhythmia, especially atrial fibrillation (AF) may occur.[sup][4] Pulsed wave tissue Doppler imaging (TDI) is one of the noninvasive, easy applicable and accessible methods to evaluate atrial electromechanical conduction properties.
demonstrated that advanced age is the most important factor related to the presence of atrial fibrillation in patients with ASDs before and after surgical closure.8 The degree of atrial dilation and the size of the ASD positively relate to changes in the atrial conduction time.1,4 We think that earlier closure of the defect can result in a more homogeneous and organised conduction of the atrial impulse.
P-wave dispersion (PWD) is an electrocardiographic measurement, which reflects a disparity in an atrial conduction. In a well-designed study, the authors aimed to analyze diagnostic accuracy of PWD in predicting recurrence of AF in patients with sinus rhythm restoration after external cardioversion.
Although the autonomic effect is eliminated after GP ablation, atrial autonomic intervention remodeling and neurohormonal disturbances in the atrium may also be responsible for AF vulnerability: for example, atrial natriuretic peptide, a substrate for arrhythmia, is associated with shortened atrial conduction time and effective refractory period (ERP) [21].
Prolonged atrial conduction is also a predisposing factor for the development of atrial flutter, where the mechanism for atrial arrhythmias is mainly due to the abnormal impulse conduction between the atria along interatrial pathways, primarily the Bachmann's Bundle, where atrial conduction times are increased.