atherogenesis


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Related to atherogenesis: dyslipidemia

atherogenesis

 [ath″er-o-jen´ĕ-sis]
formation of abnormal fatty or lipid masses in arterial walls. adj., adj atherogen´ic.

ath·er·o·gen·e·sis

(ath'er-ō-jen'ĕ-sis),
Formation of atheroma, important in the pathogenesis of arteriosclerosis.

atherogenesis

/ath·ero·gen·e·sis/ (-jen´ĕ-sis) formation of atheromatous lesions in arterial walls.atherogen´ic

atherogenesis

(ăth′ər-ō-jĕn′ĭ-sĭs)
n.
Formation of atheromatous deposits, especially on the innermost layer of arterial walls.

ath′er·o·gen′ic (-jĕn′ĭk) adj.
ath′er·o·gen·i′ci·ty (-jə-nĭs′ĭ-tē) n.

atherogenesis

Etymology: Gk, athere, porridge, oma, tumor, genein, to produce
the formation of subintimal plaques in the lining of arteries. atherogenic, adj.

ath·er·o·gen·e·sis

(ath'ĕr-ō-jen'ĕ-sis)
Formation of atheroma, important in the pathogenesis of arteriosclerosis.

atherogenesis

formation of atheromas in arterial walls.
References in periodicals archive ?
Cellular tests could be an appropriate model to study the early stages of atherogenesis and blood atherogenicity (Orekhov et al.
Expression of interleukin (IL)-18 and functional IL-18 receptor on human vascular endothelial cells, smooth muscle cells, and macrophages: implications for atherogenesis.
Impaired clearance of apoptotic cells promotes synergy between atherogenesis and autoimmune disease," The Journal of Experimental Medicine, vol.
According to this hypothesis, the increase in atheroma area of HL mice in the present study, associated with severe dyslipidemia was due to the inflammatory process, determined by the increased expression of CD40L with consequent generation of reactive oxygen species (ROS) and lower bioavailability of the NO, which induced an increase of LDL oxidation and atherogenesis.
Disruption of endothelial peroxisome proliferator-activated receptor g accelerates diet-induced atherogenesis in LDL receptor-null mice.
ii) Local dissociation: dissociation of pCRP into mCRP has been observed in membranes of apoptotic cells [42] and activated platelets in atherosclerotic plaques [43], representing an important interface between innate and adaptive immunity, thrombosis, and atherogenesis.
Further associations between dyslipidemic atherogenesis and RA, also relevant to chronic periodontitis, are considered under clinical associations.
Insulin resistance is known to increase atherogenesis and atherosclerotic plaque instability, resulting in greater risk for MI.
Elevated levels of triglyceride, cholesterol and LDL-C are documented as risk factors for atherogenesis.
Given the evidence that HCY and CRP are involved in atherogenesis, we hypothesized that significant progression of EBT calcium score (a measure of atherosclerotic plaque burden) is associated with higher levels of these markers.
Dyslipidemias cause atherogenesis leading to atherosclerosis which in turn greatly increases the risk of cardiovascular, cerebrovascular and peripheral vascular disease4,5.