antidiuresis

antidiuresis

 [an″te-, an″ti-di″u-re´sis]
the suppression of diuresis.

an·ti·di·u·re·sis

(an'tē-dī-yū-rē'sis),
Reduction of urinary volume.

an·ti·di·u·re·sis

(an'tē-dī-yŭr-ē'sis)
Reduction of urinary volume.

antidiuresis

the suppression of secretion of urine by the kidneys.
References in periodicals archive ?
Syndrome of inappropriate antidiuresis (SIAD) results from excessive secretion of vasopressin (antidiuretic hormone) or increased response by the kidneys to vasopressin.
Regulation of arginine vasopressin in the syndrome of inappropriate antidiuresis.
Atrazine has a potential to cause many acute health issues in humans, including muscle spasms, hypotension, antidiuresis, adrenal degeneration, and congestion of heart, lungs, and kidneys (U.
While the function of the coelacanth Aqp2-like channels remains unknown, the physiological role of the Aqp2-like channels in lungfishes is to promote antidiuresis during terrestrial estivation in a manner entirely synonymous with the arginine-vasoticin-induced regulation of AQP2 channels in the kidneys of modern tetrapods (Konno et al.
Sex differences in vasopressin V2 receptor expression and vasopressin-induced antidiuresis.
The mechanism of antidiuresis associated with the administration of hydrochlorothiazide to patients with vasopressin-resistant diabetes insipidus.
5) The rapid change of dose of anticonvulsant drugs influences the arginine-vasopressin fluid homeostasis systems and may be a contributory factor to the development of vasogenic oedema by disequilibrium owing to a syndrome of inappropriate antidiuresis.
The syndrome of inappropriate antidiuresis is a diagnosis of exclusion that can be established only if diuretic use and adrenal, thyroid, and pituitary insufficiency are excluded.
Urine osmolalities were less than maximally dilute (> 100 mOsm/L) with measurable levels of AVP, indicating antidiuresis as the proximate cause of the dilutional hyponatremia.
Often we see patients catheterized post-operatively or postpartum when they did not void when expected; often the patient did not void because of insufficient urine production due to postoperative antidiuresis or relative hypotension.
Antidiuresis is mediated by V2 receptors detectable on the basolateral membrane of principal cells in the collecting ducts (Kirk, 1988; Leite & Suki, 1990), luminal membrane cells of terminal inner medullary collecting ducts (Nonguchi et al.

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