anticholinesterases

anticholinesterases

cholinesterase-inhibiting agents that enhance neuromuscular transmission in myasthenia gravis; side-effects include increased sweating and bradycardia
References in periodicals archive ?
It acts differently from anticholinesterases and can reverse even profound blocks induced with vecuronium or rocuronium.
Following exposure, these anticholinesterases lead to three well-defined syndromes, i.
Nitrone spin trapping compound N-tert-butyl-alpha-phenylnitrone prevents seizures induced by anticholinesterases.
In conservative treatment of MG anticholinesterases (pyridostigmine), immunosuppressants and plasmapheresis can be used:
The patient had not received magnesium and was not previously taking or exposed to any anticholinesterases.
Modern treatment strategies typically comprise of anticholinesterases, antioxidants, alpha- and beta-secretase inhibitors, and N-methyl-D-aspartate (NMDA) receptor antagonists.
The cognitive benefits are not astounding and are actually little or no better than the currently prescribed anticholinesterases.
Anticholinesterases are frequently used in the treatment.
Determinants of the reversal time of competitive neuromuscular block by anticholinesterases.
The administration of anticholinesterases requires the co-administration of an anticholinergic drug to block potentially serious muscarinic effects, such as bradycardia, bronchospasm and an increase in secretions.
Clinicians and practitioners responsible for the diagnosis of anticholinesterase exposure in birds are cautioned that dehydration, commonly observed in sick wildlife, may mask the effect of anticholinesterases on plasma cholinesterase activity.