Antibiotic

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antibiotic

 [an″te-, an″ti-bi-ot´ik]
1. destructive of life.
2. a chemical substance produced by a microorganism that has the capacity, in dilute solutions, to kill other microorganisms or inhibit their growth. Antibiotics that are sufficiently nontoxic to the host are used as chemotherapeutic agents in the treatment of infectious diseases. See also antimicrobial agent.
antineoplastic a's (antitumor a's) a class of antineoplastic agents that apparently affect the function or the synthesis, or both, of nucleic acids and thus are cell cycle nonspecific. See also antineoplastic therapy.
broad-spectrum antibiotic one that is effective against a wide range of bacteria, both gram-positive and gram-negative.
β-lactam antibiotic any of a group of antibiotics, including the cephalosporins and the penicillins, whose chemical structure contains a β-lactam ring.

an·ti·bi·ot·ic

(an'tē-bī-ot'ik), Avoid the jargonistic use of the plural antibiotics when the reference is to a single drug.
1. Relating to antibiosis.
2. Prejudicial to life.
3. A soluble substance derived from a mold or bacterium that kills or inhibits the growth of other microorganisms.

antibiotic

(ăn′tĭ-bī-ŏt′ĭk, ăn′tī-)
n.
A substance, such as penicillin or erythromycin, produced by or derived from certain microorganisms, including fungi and bacteria, that can destroy or inhibit the growth of other microorganisms, especially bacteria. Antibiotics are widely used in the prevention and treatment of infectious diseases.
adj.
1. Of or relating to antibiotics.
2. Of or relating to antibiosis.
3. Destroying life or preventing the inception or continuance of life.

an′ti·bi·ot′i·cal·ly adv.

antibiotic

adjective Relating to the destruction of living things.
 

Herbal medicine
noun A herb said to kill or inhibit bacterial growth.
 
Mainstream medicine
(1) noun An agent obtained directly from a yeast or other organism and used against a bacterial infection.
(2) Any agent used to kill or reduce the growth of any infectious agent, including viruses, fungi and parasites.
 
Molecular biology
noun A substance that interferes with a particular step of cellular metabolism, causing either bactericidal or bacteriostatic inhibition; sometimes restricted to those having a natural biological origin.

antibiotic

adjective Relating to the destruction of living things noun Medtalk
1. An agent obtained directly from a yeast or other organism which is used against a bacterial infection.
2. Any agent used to kill or reduce the growth of any infectious agent, including viruses, fungi and parasites. See Drug resistance, Macrolide antibiotic, Polyene antibiotic Molecular biology A substance that interferes with a particular step of cellular metabolism, causing either bactericidal or bacteriostatic inhibition; sometimes restricted to those having a natural biological origin.

an·ti·bi·ot·ic

(an'tē-bī-ot'ik)
1. Relating to antibiosis.
2. Prejudicial to life.
3. Denotes any substance that acts against susceptible microorganisms.
4. Relating to such an action.

antibiotic

any substance produced by a microorganism that even in low concentrations can inhibit or kill other microorganisms. For example, PENICILLIN produced by the fungus Penicillium chrysogenum prevents the reproduction of many bacteria by preventing cell-wall synthesis. Antibiotics are frequently the products of secondary metabolism in that, while not of major importance, their formation presumably offers a selective advantage to the organism. The amount of antibiotic produced per gram of producer can be greatly enhanced by optimal culturing conditions and strong selection pressure over many generations. Unfortunately, most antibiotics are not lethal to viruses. Furthermore, continued use of an antibiotic against a generally susceptible strain of bacteria will favour survival of the few resistant members of the bacterial population, resulting eventually in an antibiotic-resistant strain.

Antibiotic

A chemical substance produced by a microorganism which can inhibit the growth of or kill other microorganisms.

antibiotic 

1. Pertaining to the ability to destroy or inhibit other living organisms.
2. A substance derived from a mould or bacterium, or produced synthetically, that destroys (bactericidal) or inhibits the growth (bacteriostatic) of other microorganisms and is thus used to treat infections. Some substances have a narrow spectrum of activity whereas others act against a wide range of both gram-positive and gram-negative organisms (broad-spectrum antibiotics). Antibiotics can be classified into several groups according to their mode of action on or within bacteria: (1) Drugs inhibiting bacterial cell wall synthesis, such as bacitracin, vancomycin and the β-lactams based agents (e.g. penicillin, cephalosporins (e.g. ceftazidime, ceftriaxone, cefuroxime). (2) Drugs affecting the bacterial cytoplasmic membrane, such as polymyxin B sulfate and gramicidin. (3) Drugs inhibiting bacterial protein synthesis, such as aminoglycosides (e.g. amikacin sulfate, framycetin sulfate, gentamicin, neomycin sulfate and tobramycin), tetracyclines, macrolides (e.g. erythromycin and azithromycin) and chloramphenicol. (4) Drugs inhibiting the intermediate metabolism of bacteria, such as sulfonamides (e.g. sulfacetamide sodium) and trimethoprim. (5) Drugs inhibiting bacterial DNA synthesis, such as nalixidic acid and fluoroquinolones (e.g. ciprofloxacin, levofloxacin, moxifloxacin, norfloxacin and ofloxacin). (6) Other antibiotics such as fusidic acid, the diamidines, such as propamidine isethionate and dibrompropamidine. Syn. antibacterial. See antiinflammatory drug; fusidic acid.

an·ti·bi·ot·ic

(an'tē-bī-ot'ik) Avoid the jargonistic use of the plural antibiotics when the reference is to a single drug.
Soluble substance derived from a mold or bacterium that kills or inhibits growth of other microorganisms.

Patient discussion about Antibiotic

Q. Can I stop taking my Antibiotics? The Doctor prescribed me Antibiotics for 10 days. I have been taking them for 5 days and feel better. Can I stop taking them?

A. you need to take all of your pills,if not it could come back.

Q. Why Is it Important to Not Use Antibiotics Often? Why is my doctor always so reluctant to prescribe me antibiotics?

A. Antibiotic resistance has become a serious problem in both developed and underdeveloped nations. By 1984 half of those with active tuberculosis in the United States had a strain that resisted at least one antibiotic. In certain settings, such as hospitals and some childcare locations, the rate of antibiotic resistance is so high that the usual, low-cost antibiotics are virtually useless for treatment of frequently seen infections. This leads to more frequent use of newer and more expensive compounds, which in turn leads to the rise of resistance to those drugs. A struggle to develop new antibiotics ensues to prevent losing future battles against infection. Therefore the doctors try to avoid using antibiotics when it is not necessary, and try to keep a certain limited use of these medications.

Q. Do Antibiotics cure a cold? I have a cold and a runny nose, should I take Antibiotics?

A. Taking antbiotics when you only have a cold can harm your chances of the effectiveness of using antibiotics when you have a severe problem. Your body can build up an immunity to antibiotics so it is only recommended to take them when your immune system can't fight off the infections. Most of the time, a cold just needs to run it's course , so drinking plenty of fluids and resting can allow your body to rejuvinate and fight the cold. To help prevent colds and viruses, look for products that help to maintain a good immune system like vitamin C. Aloe juice is another good product for your immune system. When we deal with stress and don't get enough rest, we cause havoc on our immune system, so prevention can be the best thing to do. Wishing you well!

More discussions about Antibiotic
References in periodicals archive ?
In the 4-day group, 32 patients completed the 4-day antibiotic therapy including 1 day of oral antibiotics (Figure 1).
Fourteen patients (27.4%) did not show any improvement in initial 48 hours of intravenous antibiotic therapy. These patients underwent appendectomy and were excluded from the study.
Even though molecular assays have been shown to reduce the time to optimal antibiotic therapy as well as length of hospital stay and cost of stay in previously published literature, in this observational study, we tested the appropriateness of a stratified approach to minimize the use of the rapid diagnostics to include only cases where early identification of GPCC is likely to impact empiric antibiotic therapy.
Thirdly, the probabilistic antibiotic therapy could have resulted in false negative culture of the second hip, though it was not reported in our cases.
Which biomarker is recommended by the SCCM to be used to help discontinue antibiotic therapy when a bacterial infection has been successfully treated?
But some studies showed that the antibiotic therapy after tonsillectomy helped in pain reduction and thus improving overall recovery.8,9 In our study, antibiotics did not show any significant benefit in reducing post-tonsillectomy pain.
The median duration of antibiotic therapy was 6 days (range, 2 to 25 days).
Nadeem Rizvi opined that delay in initiation of antibiotic therapy can increase mortality; hence one must start treatment as soon as pneumonia has been diagnosed.
Only results of those children who completed testing procedures (MST and spirometry) at the clinic within 48 hours of commencement and cessation of IV antibiotic therapy are presented in this report.
The outpatient antibiotic therapy clinic, which treated its 1,000th patient in July, was set up to spare patients from long hospital stays while still providing the clinical care they need.
After 5 days of IV therapy, he was switched to oral amoxicillin/ clavulanate, for a total of 14 days of antibiotic therapy.
Adding a course of antibiotic therapy to existing medications may help induce and maintain the remission of the inflammatory bowel disorder ulcerative colitis (UC), according to a study published in the August 2010 issue of the American Journal of Gastroenterology.

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