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Electrodiagnostic studies showed widespread denervation, reduced CMAP amplitudes in all nerves of the lower limbs and right upper limb, and normal SNAP responses, consistent with a severe, asymmetric process affecting anterior horn cells or motor axons.
Electrodiagnostic findings suggested a severe, asymmetric process affecting anterior horn cells or motor axons.
The results were most consistent with a severe, asymmetric process affecting anterior horn cells or motor axons.
Upper motor neuron lesions in stroke patients do not induce anterograde transneuronal degeneration in spinal anterior horn cells.
Clinical features and electrodiagnostic tests can help differentiate poliomyelitislike syndrome from Guillain-Barre syndrome by localizing damage primarily to motor axons, anterior horn cells, or both, with relative sparing of sensory nerves in the former, as opposed to localizing the damage to peripheral myelin or muscle in the latter (18,31,32,34).
Nonspecific immunoglobulin and plasmapheresis should be considered for patients with Guillain-Barre syndrome but are not indicated for patients with paralysis due to damage of anterior horn cells (30).
Electromyography and nerve-conduction studies (EMG/NCS) were indicative of a severe asymmetric process involving anterior horn cells and/or their axons.
EMG/NCS were indicative of a severe asymmetric process affecting anterior horn cells and/or their axons.
Analysis of the staining in the ALS samples showed that the viral DNA was localized in the cytoplasm of the anterior horn cells, which appeared by morphology to be neurons.

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