alpha-adrenergic blockers

alpha-adrenergic blockers (al´fəad´rəner´jik), drugs prescribed for hypertension that impede αreceptors, which in turn halts sympathetic autonomic nervous system action. See also agent, adrenergic blocking.
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TABLE 2 Antihypertensives and weight (3) Weight gain Weight neutral Alpha-adrenergic blockers ACE Inhibitors Beta-adrenergic blockers (atenolol, Angiotensin receptor blockers metoprolol, nadolol, propranolol) Beta-adrenergic blockers (carvedilol, nebivolol) CCBs Thiazides ACE, angiotensin-converting enzyme; CCBs, calcium channel blockers.
7 The optimization of this patient begins with alpha-adrenergic blockers (doxazosin and phenoxybenzamine), but faced with little improvement in myocardial contractility and instability of arterial pressure, magnesium sulfate is introduced starting in the acute crisis and during the surgical process up until the complete removal of the flow from the pheochromocytoma, ensuring a maximum level of magnesemia of 4.
Some medications commonly used to reduce blood pressure are diuretics, angiotension-converting enzyme inhibitors, beta-adrenergic blockers, calcium antagonists and alpha-adrenergic blockers.
Several general interventions assist in prevention of UI: preventing urinary tract infections and fecal impaction, controlling diabetes mellitus and heart failure, correcting hypercalemia, reducing diuretic fluids, avoiding physical or chemical restraints and managing medications that can cause UI (ACE inhibitors, calcium channel blockers, beta- and alpha-adrenergic agonists, alpha-adrenergic blockers, diuretics, cholinesterase inhibitors, psychotropics, narcotic analgesics and anticholinergics).
Thiazide diuretics, loop diuretics, calcium channel blockers, beta blockers, and alpha-adrenergic blockers can promote weight gain, generally by increasing appetite.
Pharmaceuticals (see Table 1) including sedatives/hypnotics, narcotics, diuretics, anticholinergics, alpha-adrenergic agonists or alpha-adrenergic blockers, and calcium channel blockers can contribute to the development and exacerbation of incontinence (Ghoniem & Hassouna, 1997).

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