allometric scaling

allometric scaling

scaling of dose rates of drugs, diet ratios to relative growth and size of each part of the animal, or each animal relative to the others.
References in periodicals archive ?
Anatomic and physiologic principles of allometric scaling.
Many of the intragenomic allometric scaling exponents (eight values exceeded 1.
Thus, allometric scaling was used to express grip strength per unit of FFM (grip strength/FFMp, where p is the appropriate scaling exponent), as described elsewhere [11-13].
It is unclear from our study whether the negative allometric scaling relationship is an evolutionary response to reduce vulnerability of neonates to predation and the thermal environment (Robbins and Robbins, 1979).
Human PK prediction takes into consideration bioavailability of a drug candidate, allometric scaling and physiologically-based PK (PBPK) modeling.
For instance, recent studies have demonstrated (1) a greater allometric scaling for gonad mass compared with brooding surfaces (Beekey & Karlson 2004), oxygen limitations (sometimes severe) in large embryo masses produced by both molluscs and arthropods (Strathmann & Strathmann 1995, Cohen & Strathmann 1996, Baeza & Fernandez 2002), intensive brood care, including active oxygen provision to embryo masses, in small- and moderately large-bodied species (Fernandez et al.
The first involves studies of single and multi-cellular algae which serves as model systems for allometric scaling laws in evolution.
Although their large body size (400-600 kg) requires a large quantity of food (Shipley 2010), moose tolerate a low quality diet, on a relative scale, because of the nutritional influence of allometric scaling (i.
All five data sets exhibited consistent fittings for the allometric scaling of leaf biomass in terms of length, and comparisons among data sets showed that the associated parameters can be considered as invariant within a given region.
EPA and its stakeholders be spending so much effort refining allometric scaling procedures, dialing back the estimation of exposure to the maximally exposed individual, and positing sophisticated nonl inear modes of action, while continuing to make the unscientific assertion that we are all equally susceptible to carcinogenesis?
Is West, Brown and Enquist's model of allometric scaling mathematically correct and biologically relevant?