airway hyper-reactivity


Also found in: Acronyms.

airway hyper-reactivity

The strong tendency, in people with ASTHMA, for the air passages to undergo a sudden and abnormal degree of narrowing under the influence of various stimuli such as inhaled pollen grains or house mite droppings, changes of temperature, emotional upset or exercise.
References in periodicals archive ?
Conclusion: Salbutamol significantly inhibited the contractile response of insulin, while montelukast did not produce statistically significant effect on insulin induced airway hyper-reactivity. So it is suggested that pretreatment of inhaled insulin with salbutamol may be beneficial in inhibiting the broncho-constriction induced by insulin.
Prophylactic use of montelukast decreases the symptoms of airway hyper-reactivity induced by variety of allergens and chemicals9.
Salbutamol shifted the concentration response curve of insulin to the right and downwards.Conclusions: Salbutamol significantly reduced the insulin mediated airway hyper-reactivity in guinea pigs suggesting that pre-treatment of inhaled insulin with salbutamol may have clinical implication in the amelioration of its potential respiratory adverse effects such as bronchoconstriction.Keywords: Histamine Inhaled insulin Oscillograph Tracheal muscle.
The objective of the study was to determine the magnitude of insulin-induced airway hyper-reactivity and its treatment with salbutamol.
Insulin concentration response curve in the presence of salbutamol was shifted to the right and downwards indicating a profound inhibitory effect of salbutamol on airway hyper-reactivity induced by insulin.
When untreated mice inhaled ragweed extract, their lungs suffered an influx of eosinophils (inflammation-inducing white blood cells), a jump in inflammatory signaling molecules, a buildup of mucin (a protein component of mucus) and an increase in airway hyper-reactivity (the tendency of air passages to suddenly constrict under stress).
Magnesium intake is inversely related to the risk of airway hyper-reactivity and wheezing.
Asthma is a chronic inflammatory disease with reversible airway obstruction and nonspecific airway hyper-reactivity. The local release of sensory neuropeptides from capsaicin-sensitive primary afferents causes motor neuron pathophysiology and airway inflammation and hyper-reactivity.
Asthma is a chronic inflammatory disease characterized by reversible airway obstruction, nonspecific airway hyper-reactivity, extensive neurogenic inflammatory responses involving local activation of in the inflammatory regions of neuropeptides which results in further neuron and inflammatory actions.
airway hyper-reactivity (asthma), bronchiectasis and bronchiolitis obliterans or reduced (restrictive lung function) with pathologies that involve the lung parenchyma (ILD), the pleura or the thoracic cage.
Airways: large airways for airway hyper-reactivity or scarring from bronchiectasis, smaller airways for air-trapping and bronchiolitis obliterans
Cough may persist for many weeks after an acute viral upper respiratory tract infection, and a positive challenge test in this circumstance may be diagnostically misleading because transient airway hyper-reactivity may develop.
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