abdominal aortic aneurysm

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abdominal aortic aneurysm

A focal aortic dilation of ≥ 50% increased in diameter (2 cm), accompanied by distension and weakened aortic wall.
Incidence is rising, from 12/105 (1951 to; 36/105 (1980); male:female ratio 2:1; results in 15,000 deaths/year (US).

Clinical findings
Pulsatile throbbing; rupture is characterised by sudden, severe abdominal pain radiating to back.
100% diagnostic accuracy with ultrasonography.

Difficult in obese subjects, or those with excess bowel gas, periaortic disease; technique does not document proximal or distal ends of aneurysm for surgery CT, MRI.
≥ 5.5 cm in diameter AAAs should be repaired; therapy of smaller AAAs is controversial.
Contraindications to elective reconstruction
MI in last 6 months, intractable angina or CHF, severe pulmonary insufficiency with dyspnea at rest, severe renal insufficiency, life expectancy of ≤ 2 years.
Treatment mortality is ± 6%; 25–40% of untreated cases rupture in 5 years, with a 90% mortality if ≥ 5 cm.
Segen's Medical Dictionary. © 2012 Farlex, Inc. All rights reserved.

abdominal aortic aneurysm

A focal aortic dilation of ≥ 50% ↑ in diameter, accompanied by distension and weakened aortic wall Epidemiology Incidence is rising 12/105–1951; 36/105–1980; ♂:♀ ratio 2:1; results in 15,000 deaths/yr–US 1988, population ± 230 x 106 Clinical Sudden, severe abdominal pain radiating to back Diagnosis 100% accuracy with Ultrasonography–Cons Difficult in obese subjects, or with excess bowel gas, periaortic disease, technique does not document proximal or distal ends of aneurysm for surgery Treatment ≥ 5 cm in diameter AAAs should be repaired; therapy of smaller AAAs is controversial Contraindications to elective reconstruction MI in last 6 months, intractable angina or CHF, severe pulmonary insufficiency with dyspnea at rest, severe renal insufficiency, life expectancy of ≤ 2 yrs Prognosis Treatment mortality is ± 6%; 25--40% of untreated cases rupture in 5 yrs, with a 90% mortality if ≥ 5 cm
McGraw-Hill Concise Dictionary of Modern Medicine. © 2002 by The McGraw-Hill Companies, Inc.

Abdominal Aortic Aneurysm

DRG Category:237
Mean LOS:10 days
Description:SURGICAL: Major Cardiovascular Procedures with Major CC
DRG Category:300
Mean LOS:4.6 days
Description:MEDICAL: Peripheral Vascular Disorder with CC

An abdominal aortic aneurysm (AAA) is a localized outpouching or dilation of the arterial wall in the latter portion of the descending segment of the aorta (infrarenal aorta). Aneurysms of the abdominal aorta occur more frequently than those of the thoracic aorta. AAAs are the most common type of arterial aneurysms, occurring in 3% to 10% of people older than 50 years of age in the United States. AAA may be fusiform (spindle-shaped) or saccular (pouchlike) in shape. A fusiform aneurysm in which the dilated area encircles the entire aorta is most common. A saccular aneurysm has a dilated area on only one side of the vessel.

The outpouching of the wall of the aorta occurs when the musculoelastic middle layer or media of the artery becomes weak (often caused by plaque and cholesterol deposits) and degenerative changes occur. The inner and outer layers of the arterial wall are stretched, and as the pulsatile force of the blood rushes through the aorta, the vessel wall becomes increasingly weak and the aneurysm enlarges. Abdominal aneurysms can be fatal. More than half of people with untreated aneurysms die of aneurysm rupture within 2 years.


Most authorities believe that the most common cause of AAA is atherosclerosis, which is one of several degenerative processes that can lead to the condition. The atherosclerotic process causes the buildup of plaque, which alters the integrity of the aortic wall. Ninety percent of AAAs are believed to be degenerative in origin; 5% are inflammatory. Other causes include high blood pressure, heredity, connective tissue disorders, trauma, and infections (syphilis, tuberculosis, and endocarditis). Smoking is also a contributing cause.

Genetic considerations

It is highly likely that there are genetic factors that make one susceptible to AAA, with observations of both autosomal dominant and recessive inheritance patterns. Recent work has provided evidence for genetic heterogeneity and the presence of susceptibility loci for AAA on chromosomes 19q13, 9p21, and 4q31. Family clustering of AAAs has been noted in 15% to 25% of patients undergoing surgery for AAA. In addition, AAAs are seen in rare genetic diseases such as Ehlers-Danlos syndrome or Marfan’s syndrome.

Gender, ethnic/racial, and life span considerations

Abdominal aneurysms are far more common in hypertensive men than women; from three to eight times as many men as women develop AAA. Estimates are that they are found in 2% of all men over age 55 and that they are 3.5 times more common in white men than people of other ancestries. The incidence of AAA increases with age. The occurrence is rare before age 50 and is common between the ages of 60 and 80, when the atherosclerotic process tends to become more pronounced. Ethnicity and race have no known effects on the risk for AAAs.

Global health considerations

The incidence of AAAs is 4% to 8% in men and 1% in women who live in Asia and Europe.



Seventy-five percent of AAAs are asymptomatic and are found incidentally. When the aorta enlarges and compresses the surrounding structures, patients may describe flank and back pain, epigastric discomfort, or altered bowel elimination. The pain may be deep and steady with no change if the patient shifts position. If the patient reports severe back and abdominal pain, rupture of the AAA may be imminent.

Physical examination

Inspect the patient’s abdomen for a pulsating abdominal mass in the periumbilical area, slightly to the left of midline. Auscultate over the pulsating area for an audible bruit. Gently palpate the area to determine the size of the mass and whether tenderness is present.

Watch for signs that may indicate impending aneurysm rupture, such as syncope (transient loss of consciousness and postural tone). Note subtle changes, such as a change in the characteristics and quality of peripheral pulses, changes in neurological status, and changes in vital signs such as a drop in blood pressure, increased pulse, and increased respirations. An abdominal aneurysm can impair flow to the lower extremities and cause what are known as the five Ps of ischemia: pain, pallor, pulselessness, paresthesias, and paralysis.

Because emergency surgery is indicated for both a rupture and a threatened rupture, serial and thorough assessments are important. When the aneurysm ruptures into the retroperitoneal space, hemorrhage is confined by surrounding structures, preventing immediate death by loss of blood. Examine the patient for signs of shock, including decreased capillary refill, increased pulse and respirations, a drop in urine output, weak peripheral pulses, and cool and clammy skin. When the rupture occurs anteriorly into the peritoneal cavity, rapid hemorrhage generally occurs. The patient’s vital signs and vital functions diminish rapidly. Death is usually imminent because of the rapidity of events.


In most cases, the patient with an AAA faces hospitalization, a serious surgical procedure, a stay in an intensive care unit, and a substantial recovery period. Therefore, assess the patient’s coping mechanisms and existing support systems. Assess the patient’s and significant others’ anxiety levels regarding surgery and the recovery process.

Diagnostic highlights

General Comments: Because this condition causes no symptoms, it is often diagnosed through routine physical examinations or abdominal x-rays.

TestNormal ResultAbnormality With ConditionExplanation
Standard test: abdominal ultrasonography for initial diagnosisNormal aortic diameter (2 cm diameter)Widened aorta > 3 cm in diameterDetermines presence of dilation of the infrarenal aorta
Computed tomography (CT) scanNormal aortic diameter (2 cm diameter)Locates outpouching within the aortic wall; widened aorta > 3 cmReliable assessment of size and location of aneurysm; aortic diameter exceeds 3 cm but usually will not rupture until > 5 cm; contrast-enhanced CT shows arterial anatomy; intra-aortic computed tomography angiography (IA-CTA) allows for visualization of the Adamkiewicz artery, which is important for spinal cord perfusion
Abdominal x-rayNegative studyMay show location of aneurysm with an “eggshell” appearance; AAA is evident by curvilinear calcification in the anterior wall of the aorta, displaced significantly anterior from the vertebraeAssesses size and location of aneurysm; aortic wall calcification is only seen 50% of the time (low sensitivity)

Other Tests: Magnetic resonance, aortography

Primary nursing diagnosis


Risk for fluid volume deficit related to hemorrhage


Fluid balance; Circulation status; Cardiac pump effectiveness; Hydration


Bleeding reduction; Fluid resuscitation; Blood product administration; Intravenous therapy; Circulatory care; Shock management

Planning and implementation


The treatment of choice for AAA 5.5 cm or greater in size is surgical repair. When aneurysms are smaller, some controversy exists regarding treatment. Some authorities suggest the smaller aneurysm should just be evaluated frequently by ultrasound examination or a CT scan, with surgical intervention only if the aneurysm expands. There is increasing evidence suggesting that a beta blockade, particularly propranolol, may decrease the rate of AAA expansion, and blood pressure control and smoking cessation are important. Other experts suggest elective surgical repair regardless of aneurysm size. If the aneurysm is leaking or about to rupture, immediate surgical intervention is required to improve survival rates. Approximately 50% of people with an aortic rupture do not survive.

The type and extent of surgery depend on the location of the aneurysm. Typically, an abdominal incision is made, the aneurysm is opened, clots and debris are removed, and a synthetic graft is inserted within the natural arterial wall and then sutured. During this procedure, the aorta is cross-clamped proximally and distally to the aneurysm to allow the graft to take hold. The patient is treated with heparin during the procedure to decrease the clotting of pooled blood in the lower extremities.

Patients will typically spend 2 to 3 days in the intensive care setting until their condition stabilizes. Monitor their cardiac and circulatory status closely and pay particular attention to the presence or absence of peripheral pulses and the temperature and color of the feet. Immediately report to the physician any absent or diminished pulse or cool, pale, mottled, or painful extremity. These signs could indicate an obstructed graft. Ventricular dysrhythmias are common in the postoperative period because of hypoxemia (deficient oxygen in the blood), hypothermia (temperature drop), and electrolyte imbalances. An endotracheal tube may be inserted to support ventilation. An arterial line, central venous pressure line, and peripheral intravenous lines are all typically ordered to maintain and monitor fluid balance. Adequate blood volume is supported to ensure patency of the graft and to prevent clotting of the graft as a result of low blood flow. Foley catheters are also used to assist with urinary drainage as well as with accurate intake and output measurements. Monitor for signs of infection; watch for temperature and white blood cell count elevations. Observe the abdominal wound closely, noting poor wound approximation, redness, swelling, drainage, or odor. Also report pain, tenderness, and redness in the calf of the patient’s leg. These symptoms may indicate thrombophlebitis from clot formation. If the patient develops severe postoperative back pain, notify the surgeon immediately; pain may indicate that a graft is tearing. Myocardial infarction is the most common complication.

experimental therapy.
Some medical centers are using an experimental graft that is inserted through a groin artery into the area of the aneurysm. Intravascular stents covered with prosthetic graft material such as Dacron are expandable and carry blood past the weakened portion of the aneurysm. The procedure can be performed without extensive surgery, and although it is in limited use, patients have had positive short-term results.

Pharmacologic highlights

Medication or Drug ClassDosageDescriptionRationale
Morphine1–10 mg IVOpioid analgesicRelieves surgical pain
Fentanyl50–100 mcg IVOpioid analgesicRelieves surgical pain
Antihypertensives and/or diureticsVaries by drugVaries by drugRising blood pressure may stress graft suture lines

If surgical treatment is contraindicated or not required, evidence exists that the following drugs may decrease the risk of AAA expansion or reduce the potential for complications: statins, antiplatelet therapy, angiotensin-converting enzyme inhibitors, roxithromycin, and doxycycline.


Teach the patient about the disease process, breathing and leg exercises, the surgical procedure, and postoperative routines. Support the patient by encouraging him or her to share fears, questions, and concerns. When appropriate, include support persons in the discussions. Note that the surgical procedure may be performed on an emergency basis, which limits the time available for preoperative instruction. If the patient is admitted in shock, support airway, breathing, and circulation, and expedite the surgical procedure.

Keep the incision clean and dry. Inspect the dressing every hour to check for bleeding. Use sterile techniques for all dressing changes. To ensure adequate respiratory function and to prevent complications, assist the patient with coughing and deep breathing after extubation. Splint the incision with pillows, provide adequate pain relief prior to coughing sessions, and position the patient with the head of the bed elevated to facilitate coughing. Turn the patient side to side every 2 hours to promote good ventilation and to limit skin breakdown.

Remember that emergency surgery is a time of extreme anxiety for both the patient and the significant others. Answer all questions, provide emotional support, and explain all procedures carefully. If the patient or family is not able to cope effectively, you may need to refer them for counseling.

Evidence-Based Practice and Health Policy

Greenblatt, D.Y., Greenberg, C.C., Kind, A.J., Havlena, J.A., Mell, M.W., Nelson, M.T., …Kent, K.C. (2012). Causes and implications of readmission after abdominal aortic aneurysm repair. Annals of Surgery, 256(4), 595–605.

  • Among a sample of 2,481 patients who underwent AAA repair, 30-day readmission rates were 13.3% for endovascular repairs and 12.8% for open repairs. Wound complication was the most frequent reason for readmission following both types of repairs. Additionally, readmission for graft complication was common following endovascular repair, and readmission for bowel obstruction was common following open repair.
  • Postoperative events and prolonged lengths of stay after surgical repair of more than 7 days (75th percentile) were predictive of readmission post AAA repair and associated with increased mortality.
  • Improved coordination of care across inpatient, transitional care, and outpatient settings with active surveillance for procedure-specific postoperative complications may assist in preventing early readmissions, enhance long-term survival, and reduce healthcare costs.

Documentation guidelines

  • Location, intensity, and frequency of pain and the factors that relieve pain
  • Appearance of abdominal wound (color, temperature, intactness, drainage)
  • Evidence of stability of vital signs, hydration status, bowel sounds, electrolytes
  • Presence of complications: Hypotension, graft occlusion, hypertension, cardiac dysrhythmias, infection, low urine output, thrombophlebitis, changes in consciousness, aneurysm rupture, excessive anxiety, poor wound healing

Discharge and home healthcare guidelines

wound care.
Explain the need to keep the surgical wound clean and dry. Teach the patient to observe the wound and report to the physician/practitioner any increased swelling, redness, drainage, odor, or separation of the wound edges. Also instruct the patient to notify the physician if a fever develops.

activity restrictions.
Instruct the patient to lift nothing heavier than 5 pounds for about 6 to 12 weeks and to avoid driving until her or his physician/practitioner permits. Braking while driving may increase intra-abdominal pressure and disrupt the suture line. Most surgeons temporarily discourage activities that require pulling, pushing, or stretching, such as vacuuming, changing sheets, playing tennis and golf, mowing grass, and chopping wood.

smoking cessation.
Encourage the patient to stop smoking and to attend smoking-cessation classes. Smoking-cessation materials are available through the Agency for Healthcare Research and Quality (http://www.ahrq.gov/) or the National Institute on Drug Abuse (http://www.nida.nih.gov/).

complications following surgery.
Discuss with the patient the possibility of clot formation or graft blockage. Symptoms of a clot may include pain or tenderness in the calf, and these symptoms may be accompanied by redness and warmth in the calf. Signs of graft blockage include a diminished or absent pulse and a cool, pale extremity. Tell patients to report such signs to the physician immediately.

complications for patients not requiring surgery.
Compliance with the regime of monitoring the size of the aneurysm by CT over time is essential. The patient needs to understand the prescribed medication to control hypertension. Advise the patient to report abdominal fullness or back pain, which may indicate a pending rupture.

Diseases and Disorders, © 2011 Farlex and Partners

Patient discussion about abdominal aortic aneurysm

Q. Is there a good screening test for aortic abdominal aneurysm? A friend of mine was diagnosed with an aortic abdominal aneurysm. I am afraid i might have this condition too. is there any screening test that is good for me?

A. Today there are "mobile" testing centers that charge to use ultrasound technology to detect such things as AAA. I would highly recommend it only because it can act as a preventative measure. I am 50 years old and just suffered a ruptured AAA that very nearly killed me. I was the fortunate one. This very possibly could have detected it before it actually ruptured. You may want to check in your local areas for these mobile testing centers.

More discussions about abdominal aortic aneurysm
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References in periodicals archive ?
IgE actions on CD4+ T cells, mast cells, and macrophages participate in the pathogenesis of experimental abdominal aortic aneurysms. EMBO Mol Med 2014; 6: 952-69.
Friesen, "Risk factors for abdominal aortic aneurysm: Results of a case-control study," American Journal of Epidemiology, vol.
One study in an Asian population found that infected abdominal aortic aneurysms accounted for 13.6% of cases and were associated with morbidity and mortality rates of 21%-44% [2].
Daugherty, "Prolonged infusion of angiotensin II in apoEmice promotes macrophage recruitment with continued expansion of abdominal aortic aneurysm," American Journal of Pathology, vol.
Abdominal aortic aneurysm (AAA) is an important cause of acute death hence delay in diagnosis may be fatal1-4.
Turton et al., "Twenty-year review of abdominal aortic aneurysm screening in men in the county of Gloucestershire, United Kingdom," Journal of Vascular Surgery, vol.
Quick, "Epidemiology and potential for prevention of abdominal aortic aneurysm," British Journal of Surgery, vol.
Aorto-enteric fistula after endovascular abdominal aortic aneurysm repair: case report and review.
The incidence rate of abdominal aortic aneurysm is found to be increasing worldwide due to changes in lifestyle.
incidence of primary abdominal aortic aneurysm (AAA): only 2%], leads to poor prognosis and high mortality rate.1 In general, aneurysm locally dilates normal artery by over 50% in the diameter.
Obstructive ARF caused by an inflammatory abdominal aortic aneurysm. Am J Kidney Dis 2003; 41: E9