vasovagal syncope

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Related to Vagal response: vagus nerve, Vagal episode, Vasovagal Syndrome

syncope

 [sing´kah-pe]
faint. adj. adj syn´copal, syncop´ic.
cardiac syncope sudden loss of consciousness, either with momentary premonitory symptoms or without warning, due to cerebral anemia caused by ventricular asystole, extreme bradycardia, or ventricular fibrillation.
laryngeal syncope tussive syncope.
neurocardiogenic syncope a particularly serious type of vasovagal attack; the etiology is unknown.
stretching syncope syncope associated with stretching the arms upward with the spine extended.
swallow syncope syncope associated with swallowing, a disorder of atrioventricular conduction mediated by the vagus nerve.
tussive syncope brief loss of consciousness associated with paroxysms of coughing.
vasovagal syncope vasovagal attack.

va·so·de·pres·sor syn·co·pe

faintness or loss of consciousness due to reflex reduction in blood pressure.
Synonym(s): vasovagal syncope

vasovagal syncope

a sudden loss of consciousness resulting from cerebral ischemia, secondary to decreased cardiac output, peripheral vasodilation, and bradycardia and associated with vagal activity. The condition may be triggered by pain, fright, or trauma and is accompanied by symptoms of nausea, pallor, and perspiration. Also called vasodepressor syncope.

neurocardiogenic syncope

A syncope with a psychogenic substrate and predisposition to bradycardia, hypotension and peripheral vasodilation.
 
Clinical findings
Abrupt loss of vascular tone; nausea; diaphoresis; pallor.
 
Diagnosis
Tilt test, in which the patient is placed on a tilt table at a 40º to 80º angle from horizontal and maintained in a motionless upright position for 10–15 minutes or more.
 
Management
Beta blockers—e.g., metoprolol, theophylline, disopyramide.

Mechanism
Uncertain; probably activation of myocardial mechanoreceptors (C fibres), with decreased efferent sympathetic tone and increased efferent parasympathetic tone.

vasovagal syncope

Neurocardiogenic syncope, see there.

va·so·va·gal syn·co·pe

(vā'sō-vā'găl sing'kŏ-pē)
Faintness or loss of consciousness due to increased vagus nerve (parasympathetic) activity.
Synonym(s): vasodepressor syncope.

vasovagal syncope

See FAINTING.

syncope

a temporary suspension of consciousness due to cerebral anemia; fainting.

cardiac syncope
sudden loss of consciousness due to cerebral anemia caused by ventricular asystole, extreme bradycardia or ventricular fibrillation.
drug-induced syncope
may result from abnormalities of cardiac rhythm, caused by treatment with digitalis, and hypotension caused by drugs such as diuretics, promazine and phenothiazine tranquilizers, and peripheral vasodilating agents.
laryngeal syncope
tussive syncope.
Stokes-Adams syncope
swallow syncope
syncope associated with swallowing, a disorder of atrioventricular conduction mediated by the vagus nerve.
tussive syncope
brief loss of consciousness associated with paroxysms of coughing.
vasovagal syncope
see vasovagal attack.
References in periodicals archive ?
The activation sequence and electrical conduction between LA and LSPV with vagal response
2] premature pacing at HRA, LAA, LSPV with vagal response (CVTS by [S.
The heart rate, the ERP, heterogeneity and inducibility of AF with vagal response.
In our study, with vagal response simultaneously, we delivered electronic stimulation at dog's cervical vagal trunk and paced at HRA, LAA, PV-LAJ, LSPVm and LSPVd sites, the heart rate was reduced (from 156 [+ or -] 34 bpm to 75 [+ or -] 34 bpm, on average), the ERP in HRA, LAA, PV-LAJ, LSPVm and LSPVd sites was significantly shorted (from 112.
Vagal response could be made easily by stimulating dog's cervical vagal trunk and the effect on heart lasted more than 10 hours in our study.
The LSPVd with less vagal response and lower inducibility of AF than the other sites, this might be the point that contributes to the increased ERP heterogeneity, and we may need to do further investigation in difference of vagal never distribution between LA, PV and within PV.
The conduction between left superior pulmonary vein and left atria with vagal response.
In our study, with vagal response and by endocardiac mapping, we found that the potentials of LA and PV at LA, PV-LAJ, LSPVm and LSPVd were overlapped with sinus rhythm, were separated by pacing at each sites mentioned above, and were separated remarkably by stimulating at the distal part of LSPV.
The unidirectional, bi-directional and decremented or blocked conducting characteristics within LSPV as well as across PV-LAJ in both with or without vagal response condition remind us that the totally isolation PV form LA in AF ablation will be reached only by blocking activation conduction both in sinus rhythm and also in pacing at PV site.