A natural log transformation [ln(X)] was applied to the following variables to normalize their distributions: uAs, toenail As, BMI, VCAM-1
, ICAM-1, MMP-9, TNF-[alpha], PAI-1, and 15-[F.
Average CT values for VCAM-1
, VEGFR-2, eNOS, and MMP-9 were calculated and normalized to that for [sz]-actin, and the normalized values were subjected to a 2−?
Gene expression of TNF-[alpha] (A), MCP-1 (B), IL-18 (C), VCAM-1
(D), IL-10 (E), IL-1b (F), MMP-9 (G), MMP-12 (H), LDLR (I), LRP-1 (J), CD-36 (K), in control native and grafted hearts, and LDE-MTX grafted hearts of the rabbits.
It has been determined that patients with elevated plasma VCAM-1
concentration and stable coronary heart disease are at higher risk of future cardiovascular events.
The aim of the present study is to search the association of ICAM-1, VCAM-1
, Cyclin D1 and Cathepsin D immunohistochemically with clinicopathological parameters in the patients diagnosed with invasive ductal breast carcinoma.
2]-microglobulin * TNF-[alpha] (a) * SAA Markers of endothelial * VCAM-1
cells/leukocyte activation * ICAM-1 and chemokines * P-selectin * MCP-1 * CD40 Ligand * MPO Markers of the * VWF thrombosis cascade * tPA * D-dimer * tPA antigen * PAI antigen * VWF:ADAMTS13 ratio * Fibrinogen Lipids, apolipoproteins, * [Lp-PLA.
Standardized curcuminoid extract (SCE) has the ability to protect HUVECs from vascular inflammation by suppressing LPS-induced expression of NF-[kappa]B, toll like receptor-4 (TLR-4), I CAM-1 and VCAM-1
(Angel-Morales et al.
If caveolin-1 is reduced, we see reduced levels of proatherogenic VCAM-1
Similar to serum inflammatory markers, markers of endothelial dysfunction were also increased at the end of the study compared to the start of the study among the control group; however, among the intervention group, the serum levels of p-selectin and VCAM-1
were decreased, though not significantly, and the serum level of ICAM-1 was significantly decreased at the end of the study.
Cytofluorimetric analysis of adhesion molecule surface expression showed a significant reduction of TNF-[alpha]-induced ICAM-1 and VCAM-1
surface expression evaluated as MFI in response to both DHZ and DHZ dimer (Figures 3(a) and 3(b)).
04), in contrast the differences in serum levels of VCAM-1
and PAI-1 were not statistically significant.
En celulas endoteliales la TNSF14 induce una respuesta inflamatoria por regulacion positiva de la produccion de IL-8, VCAM-1
e ICAM-1, lo que favorece la disfuncion endotelial, la vasooclusion y las trombosis (26, 27).